Modulation of Na + /K + - ATPase activity by triterpene 3β, 6β, 16β-trihidroxilup-20 (29)-ene (TTHL) limits the long-term secondary degeneration after traumatic brain injury in mice.

MLA

Della-Pace, Iuri Domingues, et al. “Modulation of Na + /K + - ATPase Activity by Triterpene 3β, 6β, 16β-Trihidroxilup-20 (29)-Ene (TTHL) Limits the Long-Term Secondary Degeneration after Traumatic Brain Injury in Mice.” European Journal of Pharmacology, vol. 854, July 2019, pp. 387–97. EBSCOhost, https://doi.org/10.1016/j.ejphar.2019.02.040.

APA

Della-Pace, I. D., Souza, T. L. de, Grauncke, A. C. B., Rambo, L. M., Ribeiro, L. R., Cipolatto, R. P., Severo, L., Papalia, W. L., Santos, A. R. S., Facundo, V. A., Oliveira, M. S., Furian, A. F., Fighera, M. R., & Royes, L. F. F. (2019). Modulation of Na + /K + - ATPase activity by triterpene 3β, 6β, 16β-trihidroxilup-20 (29)-ene (TTHL) limits the long-term secondary degeneration after traumatic brain injury in mice. European Journal of Pharmacology, 854, 387–397. https://doi.org/10.1016/j.ejphar.2019.02.040

Chicago

Della-Pace, Iuri Domingues, Thaíze Lopes de Souza, Ana Claudia Beck Grauncke, Leonardo Magno Rambo, Leandro Rodrigo Ribeiro, Rafael Parcianello Cipolatto, Leandro Severo, et al. 2019. “Modulation of Na + /K + - ATPase Activity by Triterpene 3β, 6β, 16β-Trihidroxilup-20 (29)-Ene (TTHL) Limits the Long-Term Secondary Degeneration after Traumatic Brain Injury in Mice.” European Journal of Pharmacology 854 (July): 387–97. doi:10.1016/j.ejphar.2019.02.040.