Frontline Science: TNF-α and GM-CSF1 priming augments the role of SOS1/2 in driving activation of Ras, PI3K-γ, and neutrophil proinflammatory responses.

MLA

Suire, Sabine, et al. “Frontline Science: TNF-α and GM-CSF1 Priming Augments the Role of SOS1/2 in Driving Activation of Ras, PI3K-γ, and Neutrophil Proinflammatory Responses.” Journal of Leukocyte Biology, vol. 106, no. 4, Oct. 2019, pp. 815–22. EBSCOhost, https://doi.org/10.1002/JLB.2HI0918-359RR.

APA

Suire, S., Baltanas, F. C., Segonds-Pichon, A., Davidson, K., Santos, E., Hawkins, P. T., & Stephens, L. R. (2019). Frontline Science: TNF-α and GM-CSF1 priming augments the role of SOS1/2 in driving activation of Ras, PI3K-γ, and neutrophil proinflammatory responses. Journal of Leukocyte Biology, 106(4), 815–822. https://doi.org/10.1002/JLB.2HI0918-359RR

Chicago

Suire, Sabine, Fernando C Baltanas, Anne Segonds-Pichon, Keith Davidson, Eugenio Santos, Phillip T Hawkins, and Len R Stephens. 2019. “Frontline Science: TNF-α and GM-CSF1 Priming Augments the Role of SOS1/2 in Driving Activation of Ras, PI3K-γ, and Neutrophil Proinflammatory Responses.” Journal of Leukocyte Biology 106 (4): 815–22. doi:10.1002/JLB.2HI0918-359RR.