P53 knockout mice are protected from cocaine-induced kindling behaviors via inhibiting mitochondrial oxidative burdens, mitochondrial dysfunction, and proapoptotic changes.

MLA

Mai, Huynh Nhu, et al. “P53 Knockout Mice Are Protected from Cocaine-Induced Kindling Behaviors via Inhibiting Mitochondrial Oxidative Burdens, Mitochondrial Dysfunction, and Proapoptotic Changes.” Neurochemistry International, vol. 124, Mar. 2019, pp. 68–81. EBSCOhost, https://doi.org/10.1016/j.neuint.2018.12.017.

APA

Mai, H. N., Sharma, N., Jeong, J. H., Shin, E.-J., Pham, D. T., Trinh, Q. D., Lee, Y. J., Jang, C.-G., Nah, S.-Y., Bing, G., & Kim, H.-C. (2019). P53 knockout mice are protected from cocaine-induced kindling behaviors via inhibiting mitochondrial oxidative burdens, mitochondrial dysfunction, and proapoptotic changes. Neurochemistry International, 124, 68–81. https://doi.org/10.1016/j.neuint.2018.12.017

Chicago

Mai, Huynh Nhu, Naveen Sharma, Ji Hoon Jeong, Eun-Joo Shin, Duc Toan Pham, Quynh Dieu Trinh, Yu Jeung Lee, et al. 2019. “P53 Knockout Mice Are Protected from Cocaine-Induced Kindling Behaviors via Inhibiting Mitochondrial Oxidative Burdens, Mitochondrial Dysfunction, and Proapoptotic Changes.” Neurochemistry International 124 (March): 68–81. doi:10.1016/j.neuint.2018.12.017.