Sen1, the homolog of human Senataxin, is critical for cell survival through regulation of redox homeostasis, mitochondrial function, and the TOR pathway in Saccharomyces cerevisiae.

MLA

Sariki, Santhosh Kumar, et al. “Sen1, the Homolog of Human Senataxin, Is Critical for Cell Survival through Regulation of Redox Homeostasis, Mitochondrial Function, and the TOR Pathway in Saccharomyces Cerevisiae.” The FEBS Journal, vol. 283, no. 22, Nov. 2016, pp. 4056–83. EBSCOhost, https://doi.org/10.1111/febs.13917.

APA

Sariki, S. K., Sahu, P. K., Golla, U., Singh, V., Azad, G. K., & Tomar, R. S. (2016). Sen1, the homolog of human Senataxin, is critical for cell survival through regulation of redox homeostasis, mitochondrial function, and the TOR pathway in Saccharomyces cerevisiae. The FEBS Journal, 283(22), 4056–4083. https://doi.org/10.1111/febs.13917

Chicago

Sariki, Santhosh Kumar, Pushpendra Kumar Sahu, Upendarrao Golla, Vikash Singh, Gajendra Kumar Azad, and Raghuvir S Tomar. 2016. “Sen1, the Homolog of Human Senataxin, Is Critical for Cell Survival through Regulation of Redox Homeostasis, Mitochondrial Function, and the TOR Pathway in Saccharomyces Cerevisiae.” The FEBS Journal 283 (22): 4056–83. doi:10.1111/febs.13917.