Proposed mechanisms for oligonucleotide IMT504 induced diabetes reversion in a mouse model of immunodependent diabetes.

MLA

Bianchi, María S., et al. “Proposed Mechanisms for Oligonucleotide IMT504 Induced Diabetes Reversion in a Mouse Model of Immunodependent Diabetes.” American Journal of Physiology. Endocrinology and Metabolism, vol. 311, no. 2, Aug. 2016, pp. E380–95. EBSCOhost, https://doi.org/10.1152/ajpendo.00104.2016.

APA

Bianchi, M. S., Bianchi, S., Hernado-Insúa, A., Martinez, L. M., Lago, N., Libertun, C., Chasseing, N. A., Montaner, A. D., & Lux-Lantos, V. A. (2016). Proposed mechanisms for oligonucleotide IMT504 induced diabetes reversion in a mouse model of immunodependent diabetes. American Journal of Physiology. Endocrinology and Metabolism, 311(2), E380–E395. https://doi.org/10.1152/ajpendo.00104.2016

Chicago

Bianchi, María S, Stefanía Bianchi, Andrés Hernado-Insúa, Leandro M Martinez, Néstor Lago, Carlos Libertun, Norma A Chasseing, Alejandro D Montaner, and Victoria A Lux-Lantos. 2016. “Proposed Mechanisms for Oligonucleotide IMT504 Induced Diabetes Reversion in a Mouse Model of Immunodependent Diabetes.” American Journal of Physiology. Endocrinology and Metabolism 311 (2): E380–95. doi:10.1152/ajpendo.00104.2016.