Oxymatrine attenuates CCl4-induced hepatic fibrosis via modulation of TLR4-dependent inflammatory and TGF-β1 signaling pathways.

MLA

Zhao, Hong-Wei, et al. “Oxymatrine Attenuates CCl4-Induced Hepatic Fibrosis via Modulation of TLR4-Dependent Inflammatory and TGF-Β1 Signaling Pathways.” International Immunopharmacology, vol. 36, July 2016, pp. 249–55. EBSCOhost, https://doi.org/10.1016/j.intimp.2016.04.040.

APA

Zhao, H.-W., Zhang, Z.-F., Chai, X., Li, G.-Q., Cui, H.-R., Wang, H.-B., Meng, Y.-K., Liu, H.-M., Wang, J.-B., Li, R.-S., Bai, Z.-F., & Xiao, X.-H. (2016). Oxymatrine attenuates CCl4-induced hepatic fibrosis via modulation of TLR4-dependent inflammatory and TGF-β1 signaling pathways. International Immunopharmacology, 36, 249–255. https://doi.org/10.1016/j.intimp.2016.04.040

Chicago

Zhao, Hong-Wei, Zhen-Fang Zhang, Xuan Chai, Guang-Quan Li, He-Rong Cui, Hong-Bo Wang, Ya-Kun Meng, et al. 2016. “Oxymatrine Attenuates CCl4-Induced Hepatic Fibrosis via Modulation of TLR4-Dependent Inflammatory and TGF-Β1 Signaling Pathways.” International Immunopharmacology 36 (July): 249–55. doi:10.1016/j.intimp.2016.04.040.