Cite
Enhanced Uterine Contractility and Stillbirth in Mice Lacking G Protein-Coupled Receptor Kinase 6 (GRK6): Implications for Oxytocin Receptor Desensitization.
MLA
Grotegut, Chad A., et al. “Enhanced Uterine Contractility and Stillbirth in Mice Lacking G Protein-Coupled Receptor Kinase 6 (GRK6): Implications for Oxytocin Receptor Desensitization.” Molecular Endocrinology (Baltimore, Md.), vol. 30, no. 4, Apr. 2016, pp. 455–68. EBSCOhost, https://doi.org/10.1210/me.2015-1147.
APA
Grotegut, C. A., Mao, L., Pierce, S. L., Swamy, G. K., Heine, R. P., & Murtha, A. P. (2016). Enhanced Uterine Contractility and Stillbirth in Mice Lacking G Protein-Coupled Receptor Kinase 6 (GRK6): Implications for Oxytocin Receptor Desensitization. Molecular Endocrinology (Baltimore, Md.), 30(4), 455–468. https://doi.org/10.1210/me.2015-1147
Chicago
Grotegut, Chad A, Lan Mao, Stephanie L Pierce, Geeta K Swamy, R Phillips Heine, and Amy P Murtha. 2016. “Enhanced Uterine Contractility and Stillbirth in Mice Lacking G Protein-Coupled Receptor Kinase 6 (GRK6): Implications for Oxytocin Receptor Desensitization.” Molecular Endocrinology (Baltimore, Md.) 30 (4): 455–68. doi:10.1210/me.2015-1147.