Cite
STK11/LKB1 Deficiency Promotes Neutrophil Recruitment and Proinflammatory Cytokine Production to Suppress T-cell Activity in the Lung Tumor Microenvironment.
MLA
Koyama, Shohei, et al. “STK11/LKB1 Deficiency Promotes Neutrophil Recruitment and Proinflammatory Cytokine Production to Suppress T-Cell Activity in the Lung Tumor Microenvironment.” Cancer Research, vol. 76, no. 5, Mar. 2016, pp. 999–1008. EBSCOhost, https://doi.org/10.1158/0008-5472.CAN-15-1439.
APA
Koyama, S., Akbay, E. A., Li, Y. Y., Aref, A. R., Skoulidis, F., Herter-Sprie, G. S., Buczkowski, K. A., Liu, Y., Awad, M. M., Denning, W. L., Diao, L., Wang, J., Parra-Cuentas, E. R., Wistuba, I. I., Soucheray, M., Thai, T., Asahina, H., Kitajima, S., Altabef, A., … Wong, K.-K. (2016). STK11/LKB1 Deficiency Promotes Neutrophil Recruitment and Proinflammatory Cytokine Production to Suppress T-cell Activity in the Lung Tumor Microenvironment. Cancer Research, 76(5), 999–1008. https://doi.org/10.1158/0008-5472.CAN-15-1439
Chicago
Koyama, Shohei, Esra A Akbay, Yvonne Y Li, Amir R Aref, Ferdinandos Skoulidis, Grit S Herter-Sprie, Kevin A Buczkowski, et al. 2016. “STK11/LKB1 Deficiency Promotes Neutrophil Recruitment and Proinflammatory Cytokine Production to Suppress T-Cell Activity in the Lung Tumor Microenvironment.” Cancer Research 76 (5): 999–1008. doi:10.1158/0008-5472.CAN-15-1439.