The combinatorial activation of the PI3K and Ras/MAPK pathways is sufficient for aggressive tumor formation, while individual pathway activation supports cell persistence.

MLA

Thompson, Keyata N., et al. “The Combinatorial Activation of the PI3K and Ras/MAPK Pathways Is Sufficient for Aggressive Tumor Formation, While Individual Pathway Activation Supports Cell Persistence.” Oncotarget, vol. 6, no. 34, Nov. 2015, pp. 35231–46. EBSCOhost, https://doi.org/10.18632/oncotarget.6159.

APA

Thompson, K. N., Whipple, R. A., Yoon, J. R., Lipsky, M., Charpentier, M. S., Boggs, A. E., Chakrabarti, K. R., Bhandary, L., Hessler, L. K., Martin, S. S., & Vitolo, M. I. (2015). The combinatorial activation of the PI3K and Ras/MAPK pathways is sufficient for aggressive tumor formation, while individual pathway activation supports cell persistence. Oncotarget, 6(34), 35231–35246. https://doi.org/10.18632/oncotarget.6159

Chicago

Thompson, Keyata N, Rebecca A Whipple, Jennifer R Yoon, Michael Lipsky, Monica S Charpentier, Amanda E Boggs, Kristi R Chakrabarti, et al. 2015. “The Combinatorial Activation of the PI3K and Ras/MAPK Pathways Is Sufficient for Aggressive Tumor Formation, While Individual Pathway Activation Supports Cell Persistence.” Oncotarget 6 (34): 35231–46. doi:10.18632/oncotarget.6159.