Neutrophil Elastase Activates Protease-activated Receptor-2 (PAR2) and Transient Receptor Potential Vanilloid 4 (TRPV4) to Cause Inflammation and Pain.

MLA

Zhao, Peishen, et al. “Neutrophil Elastase Activates Protease-Activated Receptor-2 (PAR2) and Transient Receptor Potential Vanilloid 4 (TRPV4) to Cause Inflammation and Pain.” The Journal of Biological Chemistry, vol. 290, no. 22, May 2015, pp. 13875–87. EBSCOhost, https://doi.org/10.1074/jbc.M115.642736.

APA

Zhao, P., Lieu, T., Barlow, N., Sostegni, S., Haerteis, S., Korbmacher, C., Liedtke, W., Jimenez-Vargas, N. N., Vanner, S. J., & Bunnett, N. W. (2015). Neutrophil Elastase Activates Protease-activated Receptor-2 (PAR2) and Transient Receptor Potential Vanilloid 4 (TRPV4) to Cause Inflammation and Pain. The Journal of Biological Chemistry, 290(22), 13875–13887. https://doi.org/10.1074/jbc.M115.642736

Chicago

Zhao, Peishen, TinaMarie Lieu, Nicholas Barlow, Silvia Sostegni, Silke Haerteis, Christoph Korbmacher, Wolfgang Liedtke, Nestor N Jimenez-Vargas, Stephen J Vanner, and Nigel W Bunnett. 2015. “Neutrophil Elastase Activates Protease-Activated Receptor-2 (PAR2) and Transient Receptor Potential Vanilloid 4 (TRPV4) to Cause Inflammation and Pain.” The Journal of Biological Chemistry 290 (22): 13875–87. doi:10.1074/jbc.M115.642736.