Cite
Alisertib induces cell cycle arrest and autophagy and suppresses epithelial-to-mesenchymal transition involving PI3K/Akt/mTOR and sirtuin 1-mediated signaling pathways in human pancreatic cancer cells.
MLA
Wang, Feng, et al. “Alisertib Induces Cell Cycle Arrest and Autophagy and Suppresses Epithelial-to-Mesenchymal Transition Involving PI3K/Akt/MTOR and Sirtuin 1-Mediated Signaling Pathways in Human Pancreatic Cancer Cells.” Drug Design, Development and Therapy, vol. 9, Jan. 2015, pp. 575–601. EBSCOhost, https://doi.org/10.2147/DDDT.S75221.
APA
Wang, F., Li, H., Yan, X.-G., Zhou, Z.-W., Yi, Z.-G., He, Z.-X., Pan, S.-T., Yang, Y.-X., Wang, Z.-Z., Zhang, X., Yang, T., Qiu, J.-X., & Zhou, S.-F. (2015). Alisertib induces cell cycle arrest and autophagy and suppresses epithelial-to-mesenchymal transition involving PI3K/Akt/mTOR and sirtuin 1-mediated signaling pathways in human pancreatic cancer cells. Drug Design, Development and Therapy, 9, 575–601. https://doi.org/10.2147/DDDT.S75221
Chicago
Wang, Feng, Hai Li, Xiao-Gang Yan, Zhi-Wei Zhou, Zhi-Gang Yi, Zhi-Xu He, Shu-Ting Pan, et al. 2015. “Alisertib Induces Cell Cycle Arrest and Autophagy and Suppresses Epithelial-to-Mesenchymal Transition Involving PI3K/Akt/MTOR and Sirtuin 1-Mediated Signaling Pathways in Human Pancreatic Cancer Cells.” Drug Design, Development and Therapy 9 (January): 575–601. doi:10.2147/DDDT.S75221.