Neurophysiologic dysfunction in basal ganglia/limbic striatal and thalamocortical circuits as a pathogenetic mechanism of obsessive-compulsive disorder.

MLA

Modell, J. G., et al. “Neurophysiologic Dysfunction in Basal Ganglia/Limbic Striatal and Thalamocortical Circuits as a Pathogenetic Mechanism of Obsessive-Compulsive Disorder.” The Journal of Neuropsychiatry and Clinical Neurosciences, vol. 1, no. 1, Winter 1989, pp. 27–36. EBSCOhost, https://doi.org/10.1176/jnp.1.1.27.

APA

Modell, J. G., Mountz, J. M., Curtis, G. C., & Greden, J. F. (1989). Neurophysiologic dysfunction in basal ganglia/limbic striatal and thalamocortical circuits as a pathogenetic mechanism of obsessive-compulsive disorder. The Journal of Neuropsychiatry and Clinical Neurosciences, 1(1), 27–36. https://doi.org/10.1176/jnp.1.1.27

Chicago

Modell, J G, J M Mountz, G C Curtis, and J F Greden. 1989. “Neurophysiologic Dysfunction in Basal Ganglia/Limbic Striatal and Thalamocortical Circuits as a Pathogenetic Mechanism of Obsessive-Compulsive Disorder.” The Journal of Neuropsychiatry and Clinical Neurosciences 1 (1): 27–36. doi:10.1176/jnp.1.1.27.