Involvement of peptidyl-prolyl isomerase Pin1 in the inhibitory effect of fluvastatin on endothelin-1-induced cardiomyocyte hypertrophy.

MLA

Sakai, Satoshi, et al. “Involvement of Peptidyl-Prolyl Isomerase Pin1 in the Inhibitory Effect of Fluvastatin on Endothelin-1-Induced Cardiomyocyte Hypertrophy.” Life Sciences, vol. 102, no. 2, May 2014, pp. 98–104. EBSCOhost, https://doi.org/10.1016/j.lfs.2014.03.018.

APA

Sakai, S., Shimojo, N., Kimura, T., Tajiri, K., Maruyama, H., Homma, S., Kuga, K., Mizutani, T., Aonuma, K., & Miyauchi, T. (2014). Involvement of peptidyl-prolyl isomerase Pin1 in the inhibitory effect of fluvastatin on endothelin-1-induced cardiomyocyte hypertrophy. Life Sciences, 102(2), 98–104. https://doi.org/10.1016/j.lfs.2014.03.018

Chicago

Sakai, Satoshi, Nobutake Shimojo, Taizo Kimura, Kazuko Tajiri, Hidekazu Maruyama, Satoshi Homma, Keisuke Kuga, Taro Mizutani, Kazutaka Aonuma, and Takashi Miyauchi. 2014. “Involvement of Peptidyl-Prolyl Isomerase Pin1 in the Inhibitory Effect of Fluvastatin on Endothelin-1-Induced Cardiomyocyte Hypertrophy.” Life Sciences 102 (2): 98–104. doi:10.1016/j.lfs.2014.03.018.