ClC-3 chloride channel/antiporter defect contributes to inflammatory bowel disease in humans and mice.

MLA

Huang, Lin-Yan, et al. “ClC-3 Chloride Channel/Antiporter Defect Contributes to Inflammatory Bowel Disease in Humans and Mice.” Gut, vol. 63, no. 10, Oct. 2014, pp. 1587–95. EBSCOhost, https://doi.org/10.1136/gutjnl-2013-305168.

APA

Huang, L.-Y., He, Q., Liang, S.-J., Su, Y.-X., Xiong, L.-X., Wu, Q.-Q., Wu, Q.-Y., Tao, J., Wang, J.-P., Tang, Y.-B., Lv, X.-F., Liu, J., Guan, Y.-Y., Pang, R.-P., & Zhou, J.-G. (2014). ClC-3 chloride channel/antiporter defect contributes to inflammatory bowel disease in humans and mice. Gut, 63(10), 1587–1595. https://doi.org/10.1136/gutjnl-2013-305168

Chicago

Huang, Lin-Yan, Qing He, Si-Jia Liang, Ying-Xue Su, Li-Xiong Xiong, Qian-Qian Wu, Qin-Yan Wu, et al. 2014. “ClC-3 Chloride Channel/Antiporter Defect Contributes to Inflammatory Bowel Disease in Humans and Mice.” Gut 63 (10): 1587–95. doi:10.1136/gutjnl-2013-305168.