Transthyretin stabilization by iododiflunisal promotes amyloid-β peptide clearance, decreases its deposition, and ameliorates cognitive deficits in an Alzheimer's disease mouse model.

MLA

Ribeiro, Carlos A., et al. “Transthyretin Stabilization by Iododiflunisal Promotes Amyloid-β Peptide Clearance, Decreases Its Deposition, and Ameliorates Cognitive Deficits in an Alzheimer’s Disease Mouse Model.” Journal of Alzheimer’s Disease : JAD, vol. 39, no. 2, 2014, pp. 357–70. EBSCOhost, https://doi.org/10.3233/JAD-131355.

APA

Ribeiro, C. A., Oliveira, S. M., Guido, L. F., Magalhães, A., Valencia, G., Arsequell, G., Saraiva, M. J., & Cardoso, I. (2014). Transthyretin stabilization by iododiflunisal promotes amyloid-β peptide clearance, decreases its deposition, and ameliorates cognitive deficits in an Alzheimer’s disease mouse model. Journal of Alzheimer’s Disease : JAD, 39(2), 357–370. https://doi.org/10.3233/JAD-131355

Chicago

Ribeiro, Carlos A, Sandra Marisa Oliveira, Luis F Guido, Ana Magalhães, Gregorio Valencia, Gemma Arsequell, Maria João Saraiva, and Isabel Cardoso. 2014. “Transthyretin Stabilization by Iododiflunisal Promotes Amyloid-β Peptide Clearance, Decreases Its Deposition, and Ameliorates Cognitive Deficits in an Alzheimer’s Disease Mouse Model.” Journal of Alzheimer’s Disease : JAD 39 (2): 357–70. doi:10.3233/JAD-131355.