Mnk1 and 2 are dispensable for T cell development and activation but important for the pathogenesis of experimental autoimmune encephalomyelitis.

MLA

Gorentla, Balachandra K., et al. “Mnk1 and 2 Are Dispensable for T Cell Development and Activation but Important for the Pathogenesis of Experimental Autoimmune Encephalomyelitis.” Journal of Immunology (Baltimore, Md. : 1950), vol. 190, no. 3, Feb. 2013, pp. 1026–37. EBSCOhost, https://doi.org/10.4049/jimmunol.1200026.

APA

Gorentla, B. K., Krishna, S., Shin, J., Inoue, M., Shinohara, M. L., Grayson, J. M., Fukunaga, R., & Zhong, X.-P. (2013). Mnk1 and 2 are dispensable for T cell development and activation but important for the pathogenesis of experimental autoimmune encephalomyelitis. Journal of Immunology (Baltimore, Md. : 1950), 190(3), 1026–1037. https://doi.org/10.4049/jimmunol.1200026

Chicago

Gorentla, Balachandra K, Sruti Krishna, Jinwook Shin, Makoto Inoue, Mari L Shinohara, Jason M Grayson, Rikiro Fukunaga, and Xiao-Ping Zhong. 2013. “Mnk1 and 2 Are Dispensable for T Cell Development and Activation but Important for the Pathogenesis of Experimental Autoimmune Encephalomyelitis.” Journal of Immunology (Baltimore, Md. : 1950) 190 (3): 1026–37. doi:10.4049/jimmunol.1200026.