Molecular mechanisms of a novel β-thalassaemia mutation due to the duplication of tetranucleotide 'AGCT' at the junction IVS-II/exon 3.

MLA

Musollino, Gennaro, et al. “Molecular Mechanisms of a Novel β-Thalassaemia Mutation Due to the Duplication of Tetranucleotide ‘AGCT’ at the Junction IVS-II/Exon 3.” Annals of Hematology, vol. 91, no. 11, Nov. 2012, pp. 1695–701. EBSCOhost, https://doi.org/10.1007/s00277-012-1526-y.

APA

Musollino, G., Mastrolonardo, G., Prezioso, R., Pagano, L., Primignani, P., Carestia, C., & Lacerra, G. (2012). Molecular mechanisms of a novel β-thalassaemia mutation due to the duplication of tetranucleotide “AGCT” at the junction IVS-II/exon 3. Annals of Hematology, 91(11), 1695–1701. https://doi.org/10.1007/s00277-012-1526-y

Chicago

Musollino, Gennaro, Gabriella Mastrolonardo, Romeo Prezioso, Leonilde Pagano, Paola Primignani, Clementina Carestia, and Giuseppina Lacerra. 2012. “Molecular Mechanisms of a Novel β-Thalassaemia Mutation Due to the Duplication of Tetranucleotide ‘AGCT’ at the Junction IVS-II/Exon 3.” Annals of Hematology 91 (11): 1695–1701. doi:10.1007/s00277-012-1526-y.