Increased Th17 and regulatory T cell responses in EBV-induced gene 3-deficient mice lead to marginally enhanced development of autoimmune encephalomyelitis.

MLA

Liu, Jin-Qing, et al. “Increased Th17 and Regulatory T Cell Responses in EBV-Induced Gene 3-Deficient Mice Lead to Marginally Enhanced Development of Autoimmune Encephalomyelitis.” Journal of Immunology (Baltimore, Md. : 1950), vol. 188, no. 7, Apr. 2012, pp. 3099–106. EBSCOhost, https://doi.org/10.4049/jimmunol.1100106.

APA

Liu, J.-Q., Liu, Z., Zhang, X., Shi, Y., Talebian, F., Carl, J. W., Jr, Yu, C., Shi, F.-D., Whitacre, C. C., Trgovcich, J., & Bai, X.-F. (2012). Increased Th17 and regulatory T cell responses in EBV-induced gene 3-deficient mice lead to marginally enhanced development of autoimmune encephalomyelitis. Journal of Immunology (Baltimore, Md. : 1950), 188(7), 3099–3106. https://doi.org/10.4049/jimmunol.1100106

Chicago

Liu, Jin-Qing, Zhenzhen Liu, Xuejun Zhang, Yun Shi, Fatemeh Talebian, Joseph W Carl Jr, Chuan Yu, et al. 2012. “Increased Th17 and Regulatory T Cell Responses in EBV-Induced Gene 3-Deficient Mice Lead to Marginally Enhanced Development of Autoimmune Encephalomyelitis.” Journal of Immunology (Baltimore, Md. : 1950) 188 (7): 3099–3106. doi:10.4049/jimmunol.1100106.