Cite
Upregulation of Nox4 by hypertrophic stimuli promotes apoptosis and mitochondrial dysfunction in cardiac myocytes.
MLA
Ago, Tetsuro, et al. “Upregulation of Nox4 by Hypertrophic Stimuli Promotes Apoptosis and Mitochondrial Dysfunction in Cardiac Myocytes.” Circulation Research, vol. 106, no. 7, Apr. 2010, pp. 1253–64. EBSCOhost, https://doi.org/10.1161/CIRCRESAHA.109.213116.
APA
Ago, T., Kuroda, J., Pain, J., Fu, C., Li, H., & Sadoshima, J. (2010). Upregulation of Nox4 by hypertrophic stimuli promotes apoptosis and mitochondrial dysfunction in cardiac myocytes. Circulation Research, 106(7), 1253–1264. https://doi.org/10.1161/CIRCRESAHA.109.213116
Chicago
Ago, Tetsuro, Junya Kuroda, Jayashree Pain, Cexiong Fu, Hong Li, and Junichi Sadoshima. 2010. “Upregulation of Nox4 by Hypertrophic Stimuli Promotes Apoptosis and Mitochondrial Dysfunction in Cardiac Myocytes.” Circulation Research 106 (7): 1253–64. doi:10.1161/CIRCRESAHA.109.213116.