Cite
Protein kinase C-dependent mitochondrial translocation of proapoptotic protein Bax on activation of inducible nitric-oxide synthase in rostral ventrolateral medulla mediates cardiovascular depression during experimental endotoxemia.
MLA
Chan, Julie Y. H., et al. “Protein Kinase C-Dependent Mitochondrial Translocation of Proapoptotic Protein Bax on Activation of Inducible Nitric-Oxide Synthase in Rostral Ventrolateral Medulla Mediates Cardiovascular Depression during Experimental Endotoxemia.” Molecular Pharmacology, vol. 71, no. 4, Apr. 2007, pp. 1129–39. EBSCOhost, https://doi.org/10.1124/mol.106.031161.
APA
Chan, J. Y. H., Chang, A. Y. W., Wang, L.-L., Ou, C.-C., & Chan, S. H. H. (2007). Protein kinase C-dependent mitochondrial translocation of proapoptotic protein Bax on activation of inducible nitric-oxide synthase in rostral ventrolateral medulla mediates cardiovascular depression during experimental endotoxemia. Molecular Pharmacology, 71(4), 1129–1139. https://doi.org/10.1124/mol.106.031161
Chicago
Chan, Julie Y H, Alice Y W Chang, Ling-Lin Wang, Chen-Chun Ou, and Samuel H H Chan. 2007. “Protein Kinase C-Dependent Mitochondrial Translocation of Proapoptotic Protein Bax on Activation of Inducible Nitric-Oxide Synthase in Rostral Ventrolateral Medulla Mediates Cardiovascular Depression during Experimental Endotoxemia.” Molecular Pharmacology 71 (4): 1129–39. doi:10.1124/mol.106.031161.