Cite
RUNX3 is frequently inactivated by dual mechanisms of protein mislocalization and promoter hypermethylation in breast cancer.
MLA
Lau, Quek Choon, et al. “RUNX3 Is Frequently Inactivated by Dual Mechanisms of Protein Mislocalization and Promoter Hypermethylation in Breast Cancer.” Cancer Research, vol. 66, no. 13, July 2006, pp. 6512–20. EBSCOhost, https://doi.org/10.1158/0008-5472.CAN-06-0369.
APA
Lau, Q. C., Raja, E., Salto-Tellez, M., Liu, Q., Ito, K., Inoue, M., Putti, T. C., Loh, M., Ko, T. K., Huang, C., Bhalla, K. N., Zhu, T., Ito, Y., & Sukumar, S. (2006). RUNX3 is frequently inactivated by dual mechanisms of protein mislocalization and promoter hypermethylation in breast cancer. Cancer Research, 66(13), 6512–6520. https://doi.org/10.1158/0008-5472.CAN-06-0369
Chicago
Lau, Quek Choon, Erna Raja, Manuel Salto-Tellez, Qiang Liu, Kosei Ito, Masafumi Inoue, Thomas Choudary Putti, et al. 2006. “RUNX3 Is Frequently Inactivated by Dual Mechanisms of Protein Mislocalization and Promoter Hypermethylation in Breast Cancer.” Cancer Research 66 (13): 6512–20. doi:10.1158/0008-5472.CAN-06-0369.