Modulation of toll-like receptor 4 expression on human monocytes by tumor necrosis factor and interleukin-6: tumor necrosis factor evokes lipopolysaccharide hyporesponsiveness, whereas interleukin-6 enhances lipopolysaccharide activity.

MLA

Tamandl, Dietmar, et al. “Modulation of Toll-like Receptor 4 Expression on Human Monocytes by Tumor Necrosis Factor and Interleukin-6: Tumor Necrosis Factor Evokes Lipopolysaccharide Hyporesponsiveness, Whereas Interleukin-6 Enhances Lipopolysaccharide Activity.” Shock (Augusta, Ga.), vol. 20, no. 3, Sept. 2003, pp. 224–29. EBSCOhost, https://doi.org/10.1097/00024382-200309000-00005.

APA

Tamandl, D., Bahrami, M., Wessner, B., Weigel, G., Ploder, M., Fürst, W., Roth, E., Boltz-Nitulescu, G., & Spittler, A. (2003). Modulation of toll-like receptor 4 expression on human monocytes by tumor necrosis factor and interleukin-6: tumor necrosis factor evokes lipopolysaccharide hyporesponsiveness, whereas interleukin-6 enhances lipopolysaccharide activity. Shock (Augusta, Ga.), 20(3), 224–229. https://doi.org/10.1097/00024382-200309000-00005

Chicago

Tamandl, Dietmar, Minu Bahrami, Barbara Wessner, Günter Weigel, Martin Ploder, Walter Fürst, Erich Roth, Georg Boltz-Nitulescu, and Andreas Spittler. 2003. “Modulation of Toll-like Receptor 4 Expression on Human Monocytes by Tumor Necrosis Factor and Interleukin-6: Tumor Necrosis Factor Evokes Lipopolysaccharide Hyporesponsiveness, Whereas Interleukin-6 Enhances Lipopolysaccharide Activity.” Shock (Augusta, Ga.) 20 (3): 224–29. doi:10.1097/00024382-200309000-00005.