Accumulation of proteolytic fragments of mutant presenilin 1 and accelerated amyloid deposition are co-regulated in transgenic mice.

MLA

Borchelt, David R., et al. “Accumulation of Proteolytic Fragments of Mutant Presenilin 1 and Accelerated Amyloid Deposition Are Co-Regulated in Transgenic Mice.” Neurobiology of Aging, vol. 23, no. 2, Mar. 2002, pp. 171–77. EBSCOhost, https://doi.org/10.1016/s0197-4580(01)00280-9.

APA

Borchelt, D. R., Lee, M. K., Gonzales, V., Slunt, H. H., Ratovitski, T., Jenkins, N. A., Copeland, N. G., Price, D. L., & Sisodia, S. S. (2002). Accumulation of proteolytic fragments of mutant presenilin 1 and accelerated amyloid deposition are co-regulated in transgenic mice. Neurobiology of Aging, 23(2), 171–177. https://doi.org/10.1016/s0197-4580(01)00280-9

Chicago

Borchelt, David R, Michael K Lee, Victoria Gonzales, Hilda H Slunt, Tamara Ratovitski, Nancy A Jenkins, Neal G Copeland, Donald L Price, and Sangram S Sisodia. 2002. “Accumulation of Proteolytic Fragments of Mutant Presenilin 1 and Accelerated Amyloid Deposition Are Co-Regulated in Transgenic Mice.” Neurobiology of Aging 23 (2): 171–77. doi:10.1016/s0197-4580(01)00280-9.