Polymorphisms in human apolipoprotein(a) kringle IV-10 and coronary artery disease: relationship to allele size, plasma lipoprotein(a) concentration, and lysine binding site activity.

MLA

Simó, J. M., et al. “Polymorphisms in Human Apolipoprotein(a) Kringle IV-10 and Coronary Artery Disease: Relationship to Allele Size, Plasma Lipoprotein(a) Concentration, and Lysine Binding Site Activity.” Journal of Molecular Medicine (Berlin, Germany), vol. 79, no. 5–6, June 2001, pp. 294–99. EBSCOhost, https://doi.org/10.1007/s001090100203.

APA

Simó, J. M., Joven, J., Vilella, E., Ribas, M., Figuera, L., Virgos, C., Sundaram, I. M., & Hoover-Plow, J. (2001). Polymorphisms in human apolipoprotein(a) kringle IV-10 and coronary artery disease: relationship to allele size, plasma lipoprotein(a) concentration, and lysine binding site activity. Journal of Molecular Medicine (Berlin, Germany), 79(5–6), 294–299. https://doi.org/10.1007/s001090100203

Chicago

Simó, J M, J Joven, E Vilella, M Ribas, L Figuera, C Virgos, I M Sundaram, and J Hoover-Plow. 2001. “Polymorphisms in Human Apolipoprotein(a) Kringle IV-10 and Coronary Artery Disease: Relationship to Allele Size, Plasma Lipoprotein(a) Concentration, and Lysine Binding Site Activity.” Journal of Molecular Medicine (Berlin, Germany) 79 (5–6): 294–99. doi:10.1007/s001090100203.