The Gly571Arg mutation, associated with the autonomic and sensory disorder congenital insensitivity to pain with anhidrosis, causes the inactivation of the NTRK1/nerve growth factor receptor.

MLA

Greco, A., et al. “The Gly571Arg Mutation, Associated with the Autonomic and Sensory Disorder Congenital Insensitivity to Pain with Anhidrosis, Causes the Inactivation of the NTRK1/Nerve Growth Factor Receptor.” Journal of Cellular Physiology, vol. 182, no. 1, Jan. 2000, pp. 127–33. EBSCOhost, https://doi.org/10.1002/(SICI)1097-4652(200001)182:1<127::AID-JCP14>3.0.CO;2-0.

APA

Greco, A., Villa, R., Fusetti, L., Orlandi, R., & Pierotti, M. A. (2000). The Gly571Arg mutation, associated with the autonomic and sensory disorder congenital insensitivity to pain with anhidrosis, causes the inactivation of the NTRK1/nerve growth factor receptor. Journal of Cellular Physiology, 182(1), 127–133. https://doi.org/10.1002/(SICI)1097-4652(200001)182:1<127::AID-JCP14>3.0.CO;2-0

Chicago

Greco, A, R Villa, L Fusetti, R Orlandi, and M A Pierotti. 2000. “The Gly571Arg Mutation, Associated with the Autonomic and Sensory Disorder Congenital Insensitivity to Pain with Anhidrosis, Causes the Inactivation of the NTRK1/Nerve Growth Factor Receptor.” Journal of Cellular Physiology 182 (1): 127–33. doi:10.1002/(SICI)1097-4652(200001)182:1<127::AID-JCP14>3.0.CO;2-0.