Overproduction of Cu/Zn-superoxide dismutase or Bcl-2 prevents the brain mitochondrial respiratory dysfunction induced by glutathione depletion.

MLA

Mérad-Saïdoune, M., et al. “Overproduction of Cu/Zn-Superoxide Dismutase or Bcl-2 Prevents the Brain Mitochondrial Respiratory Dysfunction Induced by Glutathione Depletion.” Experimental Neurology, vol. 158, no. 2, Aug. 1999, pp. 428–36. EBSCOhost, https://doi.org/10.1006/exnr.1999.7108.

APA

Mérad-Saïdoune, M., Boitier, E., Nicole, A., Marsac, C., Martinou, J. C., Sola, B., Sinet, P. M., & Ceballos-Picot, I. (1999). Overproduction of Cu/Zn-superoxide dismutase or Bcl-2 prevents the brain mitochondrial respiratory dysfunction induced by glutathione depletion. Experimental Neurology, 158(2), 428–436. https://doi.org/10.1006/exnr.1999.7108

Chicago

Mérad-Saïdoune, M, E Boitier, A Nicole, C Marsac, J C Martinou, B Sola, P M Sinet, and I Ceballos-Picot. 1999. “Overproduction of Cu/Zn-Superoxide Dismutase or Bcl-2 Prevents the Brain Mitochondrial Respiratory Dysfunction Induced by Glutathione Depletion.” Experimental Neurology 158 (2): 428–36. doi:10.1006/exnr.1999.7108.