Nε-carboxymethyllysine-mediated endoplasmic reticulum stress promotes endothelial cell injury through Nox4/MKP-3 interaction.

MLA

Lee, Wen-Jane, et al. “Nε-Carboxymethyllysine-Mediated Endoplasmic Reticulum Stress Promotes Endothelial Cell Injury through Nox4/MKP-3 Interaction.” Free Radical Biology & Medicine, vol. 74, Sept. 2014, pp. 294–306. EBSCOhost, https://doi.org/10.1016/j.freeradbiomed.2014.06.015.

APA

Lee, W.-J., Huey-Herng Sheu, W., Liu, S.-H., Yi, Y.-C., Chen, W.-C., Lin, S.-Y., Liang, K.-W., Shen, C.-C., Yeh, H.-Y., Lin, L.-Y., Tsai, Y.-C., Tien, H.-R., Lee, M.-R., Yang, T.-J., & Sheu, M.-L. (2014). Nε-carboxymethyllysine-mediated endoplasmic reticulum stress promotes endothelial cell injury through Nox4/MKP-3 interaction. Free Radical Biology & Medicine, 74, 294–306. https://doi.org/10.1016/j.freeradbiomed.2014.06.015

Chicago

Lee, Wen-Jane, Wayne Huey-Herng Sheu, Shing-Hwa Liu, Yu-Chiao Yi, Wei-Chih Chen, Shih-Yi Lin, Kae-Woei Liang, et al. 2014. “Nε-Carboxymethyllysine-Mediated Endoplasmic Reticulum Stress Promotes Endothelial Cell Injury through Nox4/MKP-3 Interaction.” Free Radical Biology & Medicine 74 (September): 294–306. doi:10.1016/j.freeradbiomed.2014.06.015.