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The p65 subunit of NF-κB involves in RIP140-mediated inflammatory and metabolic dysregulation in cardiomyocytes.

Authors :
Zhang, Luankun
Chen, Yanfang
Yue, Zhongbao
He, Yanhong
Zou, Jian
Chen, Shaorui
Liu, Min
Chen, Xi
Liu, Zhiping
Liu, Xueping
Feng, Xiaojun
Li, Min
Liu, Peiqing
Source :
Archives of Biochemistry & Biophysics. Jul2014, Vol. 554, p22-27. 6p.
Publication Year :
2014

Abstract

Highlights: [•] Elevated NF-κB activity in neonatal rat cardiomyocytes overexpressing RIP140. [•] RIP140 impacts proinflammatory response of cardiomyocytes via p65-NF-κB pathway. [•] p65-NF-κB is involved in RIP140-induced metabolic dysregulation in cardiomyocytes. [ABSTRACT FROM AUTHOR]

Details

Language :
English
ISSN :
00039861
Volume :
554
Database :
Academic Search Index
Journal :
Archives of Biochemistry & Biophysics
Publication Type :
Academic Journal
Accession number :
96582619
Full Text :
https://doi.org/10.1016/j.abb.2014.05.005