Prenatal ontogeny as a susceptibility period for cortical GABA neuron disturbances in schizophrenia.

Highlights: [•] Cortical GABA neuron abnormalities in schizophrenia may have a prenatal origin. [•] Transcription regulators and other developmental factors govern GABA neuron ontogeny. [•] Deficits in ontogenetic transcription factors have been reported in schizophrenia. [•] Maternal immune activation may be involved in GABA neuron deficits in schizophrenia. [ABSTRACT FROM AUTHOR]