Cite
A glutamate residue at the C terminus regulates activity of inward rectifier K[sup +] channels: Implication for Andersen's syndrome.
MLA
Lei Chen, et al. “A Glutamate Residue at the C Terminus Regulates Activity of Inward Rectifier K[Sup +] Channels: Implication for Andersen’s Syndrome.” Proceedings of the National Academy of Sciences of the United States of America, vol. 99, no. 12, June 2002, p. 8430. EBSCOhost, https://doi.org/10.1073/pnas.122682899.
APA
Lei Chen, Kawano, T., Bajic, S., Kaziro, Y., Itoh, H., Art, J. J., Nakajima, Y., & Nakajima, S. (2002). A glutamate residue at the C terminus regulates activity of inward rectifier K[sup +] channels: Implication for Andersen’s syndrome. Proceedings of the National Academy of Sciences of the United States of America, 99(12), 8430. https://doi.org/10.1073/pnas.122682899
Chicago
Lei Chen, Takeharu Kawano, Sibinka Bajic, Yoshito Kaziro, Hiroshi Itoh, Jonathan J. Art, Yasuko Nakajima, and Shigehiro Nakajima. 2002. “A Glutamate Residue at the C Terminus Regulates Activity of Inward Rectifier K[Sup +] Channels: Implication for Andersen’s Syndrome.” Proceedings of the National Academy of Sciences of the United States of America 99 (12): 8430. doi:10.1073/pnas.122682899.