The response to TLR ligation of human CD16+CD14− monocytes is weakly modulated as a consequence of persistent infection with the hepatitis C virus

MLA

Peng, Cheng, et al. “The Response to TLR Ligation of Human CD16+CD14− Monocytes Is Weakly Modulated as a Consequence of Persistent Infection with the Hepatitis C Virus.” Molecular Immunology, vol. 48, no. 12/13, July 2011, pp. 1505–11. EBSCOhost, https://doi.org/10.1016/j.molimm.2011.04.008.

APA

Peng, C., Liu, B.-S., de Knegt, R. J., Janssen, H. L. A., & Boonstra, A. (2011). The response to TLR ligation of human CD16+CD14− monocytes is weakly modulated as a consequence of persistent infection with the hepatitis C virus. Molecular Immunology, 48(12/13), 1505–1511. https://doi.org/10.1016/j.molimm.2011.04.008

Chicago

Peng, Cheng, Bi-Sheng Liu, Robert J. de Knegt, Harry L.A. Janssen, and André Boonstra. 2011. “The Response to TLR Ligation of Human CD16+CD14− Monocytes Is Weakly Modulated as a Consequence of Persistent Infection with the Hepatitis C Virus.” Molecular Immunology 48 (12/13): 1505–11. doi:10.1016/j.molimm.2011.04.008.