Receptor for Advanced Glycation End Product-Dependent Activation of p38 Mitogen-Activated Protein Kinase Contributes to Amyloid-β-Mediated Cortical Synaptic Dysfunction.

MLA

Origlia, Nicola, et al. “Receptor for Advanced Glycation End Product-Dependent Activation of P38 Mitogen-Activated Protein Kinase Contributes to Amyloid-β-Mediated Cortical Synaptic Dysfunction.” Journal of Neuroscience, vol. 28, no. 13, Mar. 2008, pp. 3521–30. EBSCOhost, https://doi.org/10.1523/JNEUROSCI.0204-08.2008.

APA

Origlia, N., Righi, M., Capsoni, S., Cattaneo, A., Fang Fang, Stern, D. M., Xi Chen, J., Schmidt, A. M., Arancio, O., Shi Du Yan, & Domenici, L. (2008). Receptor for Advanced Glycation End Product-Dependent Activation of p38 Mitogen-Activated Protein Kinase Contributes to Amyloid-β-Mediated Cortical Synaptic Dysfunction. Journal of Neuroscience, 28(13), 3521–3530. https://doi.org/10.1523/JNEUROSCI.0204-08.2008

Chicago

Origlia, Nicola, Massimo Righi, Simona Capsoni, Antonino Cattaneo, Fang Fang, David M. Stern, John Xi Chen, et al. 2008. “Receptor for Advanced Glycation End Product-Dependent Activation of P38 Mitogen-Activated Protein Kinase Contributes to Amyloid-β-Mediated Cortical Synaptic Dysfunction.” Journal of Neuroscience 28 (13): 3521–30. doi:10.1523/JNEUROSCI.0204-08.2008.