Irisin Ameliorate Acute Pancreatitis and Acinar Cell Viability through Modulation of the Unfolded Protein Response (UPR) and PPARγ-PGC1α-FNDC5 Pathways.

MLA

Horwitz, Avital, and Ruth Birk. “Irisin Ameliorate Acute Pancreatitis and Acinar Cell Viability through Modulation of the Unfolded Protein Response (UPR) and PPARγ-PGC1α-FNDC5 Pathways.” Biomolecules (2218-273X), vol. 14, no. 6, June 2024, p. 643. EBSCOhost, https://doi.org/10.3390/biom14060643.

APA

Horwitz, A., & Birk, R. (2024). Irisin Ameliorate Acute Pancreatitis and Acinar Cell Viability through Modulation of the Unfolded Protein Response (UPR) and PPARγ-PGC1α-FNDC5 Pathways. Biomolecules (2218-273X), 14(6), 643. https://doi.org/10.3390/biom14060643

Chicago

Horwitz, Avital, and Ruth Birk. 2024. “Irisin Ameliorate Acute Pancreatitis and Acinar Cell Viability through Modulation of the Unfolded Protein Response (UPR) and PPARγ-PGC1α-FNDC5 Pathways.” Biomolecules (2218-273X) 14 (6): 643. doi:10.3390/biom14060643.