Cite
Chronic intermittent hypoxia‐induced oxidative stress activates TRB3 and phosphorylated JNK to mediate insulin resistance and cell apoptosis in the pancreas.
MLA
Zeng, Shan, et al. “Chronic Intermittent Hypoxia‐induced Oxidative Stress Activates TRB3 and Phosphorylated JNK to Mediate Insulin Resistance and Cell Apoptosis in the Pancreas.” Clinical & Experimental Pharmacology & Physiology, vol. 51, no. 3, Mar. 2024, pp. 1–10. EBSCOhost, https://doi.org/10.1111/1440-1681.13843.
APA
Zeng, S., Wang, Y., Ai, L., Huang, L., Liu, Z., He, C., Bai, Q., & Li, Y. (2024). Chronic intermittent hypoxia‐induced oxidative stress activates TRB3 and phosphorylated JNK to mediate insulin resistance and cell apoptosis in the pancreas. Clinical & Experimental Pharmacology & Physiology, 51(3), 1–10. https://doi.org/10.1111/1440-1681.13843
Chicago
Zeng, Shan, Yeying Wang, Li Ai, Liwei Huang, Zhijuan Liu, Chunxia He, Qiaohui Bai, and Yongxia Li. 2024. “Chronic Intermittent Hypoxia‐induced Oxidative Stress Activates TRB3 and Phosphorylated JNK to Mediate Insulin Resistance and Cell Apoptosis in the Pancreas.” Clinical & Experimental Pharmacology & Physiology 51 (3): 1–10. doi:10.1111/1440-1681.13843.