Long-term iTBS Improves Neural Functional Recovery by Reducing the Inflammatory Response and Inhibiting Neuronal Apoptosis Via miR-34c-5p/p53/Bax Signaling Pathway in Cerebral Ischemic Rats.

MLA

Hu, Shouxing, et al. “Long-Term ITBS Improves Neural Functional Recovery by Reducing the Inflammatory Response and Inhibiting Neuronal Apoptosis Via MiR-34c-5p/P53/Bax Signaling Pathway in Cerebral Ischemic Rats.” Neuroscience, vol. 527, Sept. 2023, pp. 37–51. EBSCOhost, https://doi.org/10.1016/j.neuroscience.2023.07.014.

APA

Hu, S., Wang, X., Yang, X., Ouyang, S., Pan, X., Fu, Y., & Wu, S. (2023). Long-term iTBS Improves Neural Functional Recovery by Reducing the Inflammatory Response and Inhibiting Neuronal Apoptosis Via miR-34c-5p/p53/Bax Signaling Pathway in Cerebral Ischemic Rats. Neuroscience, 527, 37–51. https://doi.org/10.1016/j.neuroscience.2023.07.014

Chicago

Hu, Shouxing, Xianbin Wang, Xianglian Yang, Shuai Ouyang, Xiao Pan, Yingxue Fu, and Shuang Wu. 2023. “Long-Term ITBS Improves Neural Functional Recovery by Reducing the Inflammatory Response and Inhibiting Neuronal Apoptosis Via MiR-34c-5p/P53/Bax Signaling Pathway in Cerebral Ischemic Rats.” Neuroscience 527 (September): 37–51. doi:10.1016/j.neuroscience.2023.07.014.