Deletion of BACH1 alleviates ferroptosis and protects against LPS-triggered acute lung injury by activating Nrf2/HO-1 signaling pathway.

MLA

Wang, Rui-xuan, et al. “Deletion of BACH1 Alleviates Ferroptosis and Protects against LPS-Triggered Acute Lung Injury by Activating Nrf2/HO-1 Signaling Pathway.” Biochemical & Biophysical Research Communications, vol. 644, Feb. 2023, pp. 8–14. EBSCOhost, https://doi.org/10.1016/j.bbrc.2023.01.002.

APA

Wang, R., Gu, X., Zhang, S., Zhao, Y., Zhang, H., & Li, F. (2023). Deletion of BACH1 alleviates ferroptosis and protects against LPS-triggered acute lung injury by activating Nrf2/HO-1 signaling pathway. Biochemical & Biophysical Research Communications, 644, 8–14. https://doi.org/10.1016/j.bbrc.2023.01.002

Chicago

Wang, Rui-xuan, Xing Gu, Si-xue Zhang, Yan-jun Zhao, Hong-jun Zhang, and Fei-yan Li. 2023. “Deletion of BACH1 Alleviates Ferroptosis and Protects against LPS-Triggered Acute Lung Injury by Activating Nrf2/HO-1 Signaling Pathway.” Biochemical & Biophysical Research Communications 644 (February): 8–14. doi:10.1016/j.bbrc.2023.01.002.