Do Repeated Sprints Affect the Biceps Femoris Long Head Architecture in Football Players with and without an Injury History?—A Retrospective Study.

Simple Summary: Football is a sport which imposes an intermittent type of physical exertion, and one of the main physical demands of the game is sprinting, covering mostly short distances on repeated occasions. Sprinting imposes maximum demands, especially on the hamstrings muscle group; this activity frequently contributes to the occurrence of injury to the biceps femoris long head muscle, particularly in the later stages of the game, suggesting that fatigue may play an important role. After the injury, sprint performance and strength might be reduced, and muscle morphology can be altered even after the player returns to competition. Therefore, the aim of the study was to verify the effect of a fatiguing task on sprint performance, the biceps femoris long head muscle morphology and the hamstring muscle force capacity. Moreover, a comparison was performed between injured players and healthy controls and injured versus contralateral limbs. Muscle morphology was assessed through ultrasound techniques, while muscle force was estimated using a dynamometry assessment. It was concluded that injured players tend to be faster than the healthy control group and display shorter biceps femoris long head fascicle length when compared with the contralateral limb and healthy controls. The aim of this study was to compare the biceps femoris long head (BFlh) architecture between football players with (twelve) and without (twenty) history of BFlh injury before and after a repeated sprint task. Fascicle length (FL), pennation angle (PA) and muscle thickness (MT) were assessed at rest and in the active condition before and after the repeated sprint protocol. Athletes with previous BFlh injury showed shorter FL at rest (p = 0.014; η2p = 0.196) and active state (p < 0.001; η2p = 0.413), and greater PA at rest (p = 0.002; η2p = 0.307) and active state (p < 0.001; η2p = 0.368) before and after the task. Intra-individual comparisons showed that injured limbs have shorter FL at rest (p = 0.012; η2p = 0.519) and in the active state (p = 0.039; η2p = 0.332), and greater PA in passive (p < 0.001; η2p = 0.732) and active conditions (p = 0.018; η2p = 0.412), when compared with contralateral limbs. Injured players, at rest and in the active condition, display shorter BFlh FL and greater PA than contralateral and healthy controls after repeated sprints. Moreover, the BFlh of injured players presented a different architectural response to the protocol compared with the healthy controls. [ABSTRACT FROM AUTHOR]