Dimethyl itaconate protects against lippolysacchride-induced mastitis in mice by activating MAPKs and Nrf2 and inhibiting NF-κB signaling pathways.

MLA

Zhao, Caijun, et al. “Dimethyl Itaconate Protects against Lippolysacchride-Induced Mastitis in Mice by Activating MAPKs and Nrf2 and Inhibiting NF-ΚB Signaling Pathways.” Microbial Pathogenesis, vol. 133, Aug. 2019, p. 103541. EBSCOhost, https://doi.org/10.1016/j.micpath.2019.05.024.

APA

Zhao, C., Jiang, P., He, Z., Yuan, X., Guo, J., Li, Y., Hu, X., Cao, Y., Fu, Y., & Zhang, N. (2019). Dimethyl itaconate protects against lippolysacchride-induced mastitis in mice by activating MAPKs and Nrf2 and inhibiting NF-κB signaling pathways. Microbial Pathogenesis, 133, 103541. https://doi.org/10.1016/j.micpath.2019.05.024

Chicago

Zhao, Caijun, Peng Jiang, Zhaoqi He, Xin Yuan, Jian Guo, Yanyi Li, Xiaoyu Hu, Yongguo Cao, Yunhe Fu, and Naisheng Zhang. 2019. “Dimethyl Itaconate Protects against Lippolysacchride-Induced Mastitis in Mice by Activating MAPKs and Nrf2 and Inhibiting NF-ΚB Signaling Pathways.” Microbial Pathogenesis 133 (August): 103541. doi:10.1016/j.micpath.2019.05.024.