Abstract 14989: Excessive Protein O-GlcNAcylation Contributes to the Development of Coronary Microvascular Disease in Type 2 Diabetic Mice.

MLA

Tsuji-Hosokawa, Atsumi, et al. “Abstract 14989: Excessive Protein O-GlcNAcylation Contributes to the Development of Coronary Microvascular Disease in Type 2 Diabetic Mice.” Circulation, vol. 138, Nov. 2018, p. A14989. EBSCOhost, widgets.ebscohost.com/prod/customlink/proxify/proxify.php?count=1&encode=0&proxy=&find_1=&replace_1=&target=https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&scope=site&db=asx&AN=135765756&authtype=sso&custid=ns315887.

APA

Tsuji-Hosokawa, A., Watanabe, M., Zhang, Q., Varghese, M. V., Wang, J., & Makino, A. (2018). Abstract 14989: Excessive Protein O-GlcNAcylation Contributes to the Development of Coronary Microvascular Disease in Type 2 Diabetic Mice. Circulation, 138, A14989.

Chicago

Tsuji-Hosokawa, Atsumi, Makiko Watanabe, Qian Zhang, Mathews V Varghese, Jian Wang, and Ayako Makino. 2018. “Abstract 14989: Excessive Protein O-GlcNAcylation Contributes to the Development of Coronary Microvascular Disease in Type 2 Diabetic Mice.” Circulation 138 (November): A14989. http://widgets.ebscohost.com/prod/customlink/proxify/proxify.php?count=1&encode=0&proxy=&find_1=&replace_1=&target=https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&scope=site&db=asx&AN=135765756&authtype=sso&custid=ns315887.