Cite
The proteasome inhibitor bortezomib induces testicular toxicity by upregulation of oxidative stress, AMP-activated protein kinase (AMPK) activation and deregulation of germ cell development in adult murine testis.
MLA
Li, Wei, et al. “The Proteasome Inhibitor Bortezomib Induces Testicular Toxicity by Upregulation of Oxidative Stress, AMP-Activated Protein Kinase (AMPK) Activation and Deregulation of Germ Cell Development in Adult Murine Testis.” Toxicology & Applied Pharmacology, vol. 285, no. 2, June 2015, pp. 98–109. EBSCOhost, https://doi.org/10.1016/j.taap.2015.04.001.
APA
Li, W., Fu, J., Zhang, S., Zhao, J., Xie, N., & Cai, G. (2015). The proteasome inhibitor bortezomib induces testicular toxicity by upregulation of oxidative stress, AMP-activated protein kinase (AMPK) activation and deregulation of germ cell development in adult murine testis. Toxicology & Applied Pharmacology, 285(2), 98–109. https://doi.org/10.1016/j.taap.2015.04.001
Chicago
Li, Wei, Jianfang Fu, Shun Zhang, Jie Zhao, Nianlin Xie, and Guoqing Cai. 2015. “The Proteasome Inhibitor Bortezomib Induces Testicular Toxicity by Upregulation of Oxidative Stress, AMP-Activated Protein Kinase (AMPK) Activation and Deregulation of Germ Cell Development in Adult Murine Testis.” Toxicology & Applied Pharmacology 285 (2): 98–109. doi:10.1016/j.taap.2015.04.001.