314 results on '"van de Loo, Fons A. J."'
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2. Protective Effect of Bovine Milk Extracellular Vesicles on Alveolar Bone Loss
3. Polyethylene glycol precipitation is an efficient method to obtain extracellular vesicle-depleted fetal bovine serum
4. Influence of mesenchymal stem cell-derived extracellular vesicles in vitro and their role in ageing
5. The serum amyloid A3 promoter-driven luciferase reporter mice is a valuable tool to image early renal fibrosis development and shows the therapeutic effect of glucosyl-hesperidin treatment
6. Protective Effect of Bovine Milk Extracellular Vesicles on Alveolar Bone Loss.
7. Profiling of plasma extracellular vesicles identifies proteins that strongly associate with patient's global assessment of disease activity in rheumatoid arthritis.
8. Gas6/Axl Axis Activation Dampens the Inflammatory Response in Osteoarthritic Fibroblast-like Synoviocytes and Synovial Explants
9. Transcriptional profiling distinguishes inner and outer annulus fibrosus from nucleus pulposus in the bovine intervertebral disc
10. Regulated promoters
11. Role of IL-1 in erosive arthritis, lessons from animal models
12. Fcγ receptor-mediated influx of S100A8/A9-producing neutrophils as inducer of bone erosion during antigen-induced arthritis
13. Gene Therapy in Murine Arthritis : Cytokine-Directed Targeting
14. Early pain in females is linked to late pathological features in murine experimental osteoarthritis.
15. Identification of Transcription Factors Responsible for a Transforming Growth Factor-β-Driven Hypertrophy-like Phenotype in Human Osteoarthritic Chondrocytes
16. IL-32, a Proinflammatory Cytokine in Rheumatoid Arthritis
17. Disease-regulated local IL-10 gene therapy diminishes synovitis and cartilage proteoglycan depletion in experimental arthritis
18. Nox2 Deficiency Reduces Cartilage Damage and Ectopic Bone Formation in an Experimental Model for Osteoarthritis
19. Interleukin-21 Receptor Deficiency Increases the Initial Toll-like Receptor 2 Response but Protects Against Joint Pathology by Reducing Th1 and Th17 Cells During Streptococcal Cell Wall Arthritis
20. Flood Control: How Milk-Derived Extracellular Vesicles Can Help to Improve the Intestinal Barrier Function and Break the Gut–Joint Axis in Rheumatoid Arthritis
21. Gene therapy works in animal models of rheumatoid arthritis so what!
22. Comparing Approaches to Normalize, Quantify, and Characterize Urinary Extracellular Vesicles
23. Therapeutic Potential for Regulation of the Nuclear Factor Kappa-B Transcription Factor p65 to Prevent Cellular Senescence and Activation of Pro-Inflammatory in Mesenchymal Stem Cells
24. Toll-like Receptor 2 Controls Acute Immune Complex–Driven Arthritis in Mice by Regulating the Inhibitory Fcγ Receptor IIB
25. Toll-like receptor 4 in bone marrow-derived cells as well as tissue-resident cells participate in aggravating autoimmune destructive arthritis
26. Enhanced suppressor of cytokine signaling 3 in arthritic cartilage dysregulates human chondrocyte function
27. Destructive role of myeloid differentiation factor 88 and protective role of TRIF in interleukin-17–dependent arthritis in mice
28. IL-21 induces socs-mediated suppression of TLR triggering but aggravates TH17-driven destructive arthritis
29. NIR-fluorescence imaging points at a role for matrix-metalloproteinases in causing irreversible cartilage damage during collagen-induced arthritis
30. A pivotal role for antigen-presenting cells overexpressing SOCS3 in controlling invariant NKT cell responses during collagen-induced arthritis
31. Toll-like receptor 4 signalling is specifically TGF-beta-activated kinase 1 independent in synovial fibroblasts
32. Identifying the TLR4 bearing target cell in experimental arthritis
33. Toll-like receptor-4 signalling is specifically tak1-independent in synovial fibroblasts
34. Synergy between 15-lipoxygenase and secreted PLA 2 promotes inflammation by formation of TLR4 agonists from extracellular vesicles
35. Selective Increment of Synovial Soluble TYRO3 Correlates with Disease Severity and Joint Inflammation in Patients with Rheumatoid Arthritis
36. Bovine Milk Extracellular Vesicles Are Osteoprotective by Increasing Osteocyte Numbers and Targeting RANKL/OPG System in Experimental Models of Bone Loss
37. An optimized method for plasma extracellular vesicles isolation to exclude the copresence of biological drugs and plasma proteins which impairs their biological characterization
38. A roadmap to target interleukin-6 in osteoarthritis
39. Bovine Milk‐Derived Extracellular Vesicles Inhibit Catabolic and Inflammatory Processes in Cartilage from Osteoarthritis Patients.
40. Splenic Suppressor of Cytokine Signaling 3 Transgene Expression Affects T Cell Responses and Prevents Development of Collagen-Induced Arthritis
41. ANTICYTOKINE TREATMENT OF ESTABLISHED TYPE II COLLAGEN-INDUCED ARTHRITIS IN DBA/1 MICE: A Comparative Study using Anti-TNFα, Anti-IL-αa/β, and IL-1Ra
42. Local Activation of STAT-1 and STAT-3 in the Inflamed Synovium During Zymosan-Induced Arthritis: Exacerbation of Joint Inflammation in STAT-1 Gene–Knockout Mice
43. Growth Plate Damage, a Feature of Juvenile Idiopathic Arthritis, Can Be Induced by Adenoviral Gene Transfer of Oncostatin M: A Comparative Study in Gene-Deficient Mice
44. Increased Expression of Fcγ Receptors II and III on Macrophages of Rheumatoid Arthritis Patients Results in Higher Production of Tumor Necrosis Factor α and Matrix Metalloproteinase
45. Inducible Nitric Oxide Synthase Mediates Bone Loss in Ovariectomized Mice
46. Local Interleukin-12 Gene Transfer Promotes Conversion of an Acute Arthritis to a Chronic Destructive Arthritis
47. Genetic Ablation of Interferon-γ Up-Regulates Interleukin-1β Expression and Enables the Elicitation of Collagen-Induced Arthritis in a Nonsusceptible Mouse Strain
48. Inducible Nitric Oxide Synthase—Knockout Mice Exhibit Resistance to Pleurisy and Lung Injury Caused by Carrageenan
49. REDUCTION OF INTERLEUKIN-17–INDUCED INHIBITION OF CHONDROCYTE PROTEOGLYCAN SYNTHESIS IN INTACT MURINE ARTICULAR CARTILAGE BY INTERLEUKIN-4
50. Supplementation of diet with non-digestible oligosaccharides alters the intestinal microbiota, but not arthritis development, in IL-1 receptor antagonist deficient mice
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