335 results on '"te Riele, Hein"'
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2. HideRNAs protect against CRISPR-Cas9 re-cutting after successful single base-pair gene editing
3. ADARp150 counteracts whole genome duplication.
4. Paradoxical activation of oncogenic signaling as a cancer treatment strategy
5. Paradoxical Activation of Oncogenic Signaling as a Cancer Treatment Strategy
6. Targeting CDC7 potentiates ATR-CHK1 signaling inhibition through induction of DNA replication stress in liver cancer
7. Inducing and exploiting vulnerabilities for the treatment of liver cancer
8. Warsaw Breakage Syndrome associated DDX11 helicase resolves G-quadruplex structures to support sister chromatid cohesion
9. Effective CRISPR/Cas9-mediated correction of a Fanconi anemia defect by error-prone end joining or templated repair
10. Supplementary Information from Pretreatment microRNA Expression Impacting on Epithelial-to-Mesenchymal Transition Predicts Intrinsic Radiosensitivity in Head and Neck Cancer Cell Lines and Patients
11. Data from Pretreatment microRNA Expression Impacting on Epithelial-to-Mesenchymal Transition Predicts Intrinsic Radiosensitivity in Head and Neck Cancer Cell Lines and Patients
12. Supplementary Figure 1 and Tables 1-8 from Pretreatment microRNA Expression Impacting on Epithelial-to-Mesenchymal Transition Predicts Intrinsic Radiosensitivity in Head and Neck Cancer Cell Lines and Patients
13. Data Supplement from RB Family Tumor Suppressor Activity May Not Relate to Active Silencing of E2F Target Genes
14. Data from RB Family Tumor Suppressor Activity May Not Relate to Active Silencing of E2F Target Genes
15. LNA modification of single-stranded DNA oligonucleotides allows subtle gene modification in mismatch-repair-proficient cells
16. Paradoxical activation of oncogenic signaling as a cancer treatment strategy
17. Unexpected moves: a conformational change in MutSα enables high-affinity DNA mismatch binding
18. Truncation of the MSH2 C-terminal 60 amino acids disrupts effective DNA mismatch repair and is causative for Lynch syndrome
19. The Retinoblastoma Gene Family in Cell Cycle Regulation and Suppression of Tumorigenesis
20. Pro-mutagenic effects of the gut microbiota in a Lynch syndrome mouse model
21. Somatic genomic alterations in retinoblastoma beyond RB1 are rare and limited to copy number changes
22. Additional file 4 of Targeting CDC7 potentiates ATR-CHK1 signaling inhibition through induction of DNA replication stress in liver cancer
23. Additional file 2 of Targeting CDC7 potentiates ATR-CHK1 signaling inhibition through induction of DNA replication stress in liver cancer
24. The APC/C recruits cyclin B1-Cdk1-Cks in prometaphase before D box recognition to control mitotic exit
25. Functional analysis of MSH2 unclassified variants found in suspected Lynch syndrome patients reveals pathogenicity due to attenuated mismatch repair
26. The Widely Used Antihelmintic Drug Albendazole is a Potent Inducer of Loss of Heterozygosity
27. Msh2 deficiency does not contribute to cisplatin resistance in mouse embryonic stem cells
28. CTG repeat instability and size variation timing in DNA repair‐deficient mice
29. Retinoblastoma protein functions as a molecular switch determining white versus brown adipocyte differentiation
30. Heterozygosity for p53 promotes microsatellite instability and tumorigenesis on a Msh2 deficient background
31. Tumor formation in mice with somatic inactivation of the retinoblastoma gene in interphotoreceptor retinol binding protein-expressing cells
32. Methylation tolerance in mismatch repair proficient cells with low MSH2 protein level
33. Retinoblastoma gene-independent G1 phase arrest by flavone, phosphatidylinositol 3-kinase inhibitor, and histone deacetylase inhibitor
34. p27kip1-independent cell cycle regulation by MYC
35. Fancf-deficient mice are prone to develop ovarian tumours
36. Characterization of MSH2 variants by endogenous gene modification in mouse embryonic stem cells
37. Extensive trimming of short single-stranded DNA oligonucleotides during replication-coupled gene editing in mammalian cells
38. The RECQL helicase prevents replication fork collapse during replication stress
39. Ablation of the Retinoblastoma gene family deregulates G(sub.1) control causing immortalization and increased cell turnover under growth-restricting conditions
40. Azathioprine-Induced Carcinogenesis in Mice According to Msh2 Genotype
41. Subtle gene modification in mouse ES cells: evidence for incorporation of unmodified oligonucleotides without induction of DNA damage
42. Parameters of oligonucleotide-mediated gene modification in mouse ES cells
43. Fancm-deficient mice reveal unique features of Fanconi anemia complementation group M
44. Mitogen requirement for cell cycle progression in the absence of pocket protein activity
45. p107 is a suppressor of retinoblastoma development in pRb-deficient mice
46. Generation of a mouse mutant by oligonucleotide-mediated gene modification in ES cells
47. Msh2 deficiency increases susceptibility to benzo[a]pyrene-induced lymphomagenesis
48. Targeted gene modification in mismatch-repair-deficient embryonic stem cells by single-stranded DNA oligonucleotides
49. Mismatch repair gene Msh2 modifies the timing of early disease in HdhQ111 striatum
50. Somatic expansion behaviour of the (CTG)n repeat in myotonic dystrophy knock-in mice is differentially affected by Msh3 and Msh6 mismatch–repair proteins
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