17 results on '"Zipei Liu"'
Search Results
2. Assessing Spatiotemporal Variation of Forest Aboveground Carbon Sequestration Coupling Landscape Models With Remote Sensing Datasets In Western Sichuan, China.
- Author
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Shiyu Deng, Mingfang Zhang 0003, Yong Wang 0011, Yiping Hou, Enxu Yu, and Zipei Liu
- Published
- 2024
- Full Text
- View/download PDF
3. A Novel Fusion Architecture for PD Detection Using Semi-Supervised Speech Embeddings.
- Author
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T. M. Tariq Adnan, Abdelrahman Abdelkader, Zipei Liu, Ekram Hossain, Sooyong Park, Md. Saiful Islam 0013, and Ehsan Hoque 0001
- Published
- 2024
- Full Text
- View/download PDF
4. Assessing Effects of Climate Variability and Forest Disturbance on Annual Streamflow of the Stellako Watershed, Canada.
- Author
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Zipei Liu, Mingfang Zhang 0003, Shiyu Deng, Yiping Hou, Yali Xu, Hui Lian, and Yong Wang 0011
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- 2023
- Full Text
- View/download PDF
5. Research on Machine Learning Prediction algorithm of Financial data based on Stochastic Process Analysis and Evolutionary Algorithm.
- Author
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Jianzhe Liu and Zipei Liu
- Published
- 2021
- Full Text
- View/download PDF
6. Managing the forest-water nexus for climate change adaptation
- Author
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Mingfang Zhang, Shirong Liu, Julia Jones, Ge Sun, Xiaohua Wei, David Ellison, Emma Archer, Steve McNulty, Heidi Asbjornsen, Zhiqiang Zhang, Yusuf Serengil, Meinan Zhang, Zhen Yu, Qiang Li, Junwei Luan, Ibrahim Yurtseven, Yiping Hou, Shiyu Deng, and Zipei Liu
- Subjects
Forestry ,Management, Monitoring, Policy and Law ,Nature and Landscape Conservation - Published
- 2022
7. The Pringle manoeuvre should be avoided in hepatectomy for cancer patients due to its side effects on tumor recurrence and worse prognosis
- Author
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Xiaobin, Feng, Zipei, Liu, Shuguo, Zheng, Jiahong, Dong, and Xiaowu, Li
- Published
- 2009
- Full Text
- View/download PDF
8. Children's judgements of positive claims people make about themselves
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Gail D. Heyman, Brian J. Compton, Jamie Amemiya, and Zipei Liu
- Subjects
media_common.quotation_subject ,Developmental and Educational Psychology ,Pragmatics ,Psychology ,Social psychology ,Reputation ,media_common - Published
- 2020
9. 67 laminin receptor promotes the malignant potential of tumour cells up-regulating lysyl oxidase-like 2 expression in cholangiocarcinoma
- Author
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Peng Jiang, Jing Xu, Qiang He, Shuguang Wang, Yang Gao, Tianyu Li, Dajing Li, De-chun Wang, Zipei Liu, Xiaowu Li, and Feng Tian
- Subjects
Male ,Ribosomal Proteins ,Pathology ,medicine.medical_specialty ,Mice, Nude ,Lysyl oxidase ,digestive system ,Metastasis ,Cholangiocarcinoma ,Receptors, Laminin ,Small hairpin RNA ,Mice ,Cell Movement ,Cell Line, Tumor ,Biomarkers, Tumor ,medicine ,Animals ,Humans ,Neoplasm Invasiveness ,neoplasms ,Aged ,Gene knockdown ,Hepatology ,LOXL2 ,business.industry ,Gastroenterology ,Cell Differentiation ,Transfection ,Middle Aged ,Prognosis ,medicine.disease ,digestive system diseases ,Up-Regulation ,Survival Rate ,67 kDa Laminin Receptor ,Bile Ducts, Intrahepatic ,Bile Duct Neoplasms ,Gene Knockdown Techniques ,Lymphatic Metastasis ,Immunohistochemistry ,Female ,Amino Acid Oxidoreductases ,business - Abstract
Background 67 laminin receptor (67LR) plays an important role in the invasion and metastasis of cholangiocarcinoma, but its mechanism remains unclear. Aims We investigated the clinical significance of 67LR and its relation to lysyl oxidase-like 2 (LOXL2) in 67LR-mediated invasion and metastasis in cholangiocarcinoma. Methods The clinical significance of 67LR and LOXL2 expression and the prognosis of patients were investigated in 73 cancerous and 32 paracancerous tissues by immunohistochemistry. The impact of LOXL2 on invasion, metastasis and 67LR expression was evaluated in cholangiocarcinoma cells by shRNA or expressed-plasmid transfection. Results Expression of 67LR was recognized in 35.62% cholangiocarcinoma tissue, and none in paracancerous tissues. LOXL2 was positively correlated with expression of 67LR. Expression of 67LR or LOXL2 in cholangiocarcinomas was significantly associated with lymph node metastasis, differentiation and poor overall survival. Cox analysis showed that 67LR can act as an independent prognostic biomarker of prognosis in cholangiocarcinoma patients. Expression of LOXL2 decreased by knockdown of 67LR and increased by overexpression of 67LR in cholangiocarcinoma cells. Knockdown of LOXL2 reduced invasion and metastasis in vitro and in vivo. Conclusion 67LR may regulate the expression of LOXL2 to promote invasion and metastasis in cholangiocarcinoma cells. It could be used as an independent prognostic marker in cholangiocarcinoma patients.
- Published
- 2014
10. Aberrant expression of GATA binding protein 6 correlates with poor prognosis and promotes metastasis in cholangiocarcinoma
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Feng Tian, Fei Guo, Dajiang Li, Xiaowu Li, Shuguang Wang, Wei Liu, Jianwei Li, Lei Cai, Xin Zhao, Peng Jiang, Zipei Liu, and Jian Chen
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Male ,Ribosomal Proteins ,endocrine system ,Cancer Research ,Pathology ,medicine.medical_specialty ,Blotting, Western ,Transplantation, Heterologous ,Cell ,Mice, Nude ,Kaplan-Meier Estimate ,Biology ,Metastasis ,Cholangiocarcinoma ,Receptors, Laminin ,Small hairpin RNA ,Mice ,Cell Movement ,Cell Line, Tumor ,GATA6 Transcription Factor ,medicine ,Animals ,Humans ,Promoter Regions, Genetic ,Aged ,Proportional Hazards Models ,Gene knockdown ,GATA6 ,Reverse Transcriptase Polymerase Chain Reaction ,Transfection ,Middle Aged ,Prognosis ,medicine.disease ,Immunohistochemistry ,Transplantation ,Bile Ducts, Intrahepatic ,medicine.anatomical_structure ,Bile Duct Neoplasms ,Oncology ,Lymphatic Metastasis ,Cancer research ,Female ,RNA Interference ,Protein Binding - Abstract
Aim GATA6, a zinc-finger transcription factor, functions as a tumour promoter or suppresser according to different tumour origins. We investigated the clinical significance of GATA6 and its role in invasion and metastasis in cholangiocarcinoma (CCA). Methods Expression of GATA6 in 87 cancerous, 24 paracancerous, 32 lymph-node metastatic and 8 liver metastatic samples from 87 CCA patients undergoing surgical resection was detected by immunohistochemistry. Impact of GATA6 on invasion, metastasis and 67 kDa laminin receptor expression (67LR) was evaluated in CCA cells by shRNA lentivirus or expressed-plasmid transfection. Results Aberrant expression of GATA6 in CCAs was significantly associated with lymph-node metastasis. GATA6 expression was higher in lymph-node and liver metastatic tissues compared with primary cancerous tissues. Kaplan–Meier analysis showed GATA6 expression correlated with poor overall survival and early recurrence in CCAs. Cox analysis suggested GATA6 was an independent prognostic marker for overall survival and recurrence-free survival. CCA cell invasion and migration were decreased by GATA6 knockdown and enhanced by GATA6 overexpression in vitro . Knockdown of GATA6 reduced CCA cell metastasis by xenotransplantation into nude mice. 67LR, which is overexpressed in CCAs and promotes invasion and metastasis through several pathways, positively correlated with GATA6 expression in 87 CCAs. Both mRNA and protein levels of 67LR were regulated by GATA6 in CCA cells. Moreover, ChIP analysis showed GATA6 bound to 67LR gene promoter in CCA cells. Conclusion Aberrant expression of GATA6 correlates with poor prognosis and promotes invasion and metastasis in CCA.
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- 2013
11. Downregulation of Mucins in Graft Bile Ducts After Liver Transplantation in Rats
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Xiaowu Li, Shuguang Wang, Jing Xu, Long Cheng, Yi Zeng, Feng Tian, Zipei Liu, and Dajiang Li
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Male ,medicine.medical_specialty ,medicine.medical_treatment ,Down-Regulation ,Liver transplantation ,GATA Transcription Factors ,digestive system ,Gastroenterology ,Rats, Sprague-Dawley ,Downregulation and upregulation ,GATA6 Transcription Factor ,Internal medicine ,medicine ,Animals ,RNA, Messenger ,MUC1 ,Mucin-3 ,Transplantation ,Base Sequence ,Mucin-4 ,Bile duct ,GATA4 ,business.industry ,Cold Ischemia ,Mucin-1 ,Mucin ,Mucins ,GATA3 ,GATA4 Transcription Factor ,Liver Transplantation ,Rats ,surgical procedures, operative ,medicine.anatomical_structure ,Reperfusion Injury ,Immunohistochemistry ,Bile Ducts ,business - Abstract
BACKGROUND Nonanastomotic biliary strictures represent a serious complication after orthotopic liver transplantation (OLT). This study investigates the potential role of mucins in bile duct injury after OLT. METHODS Sprague-Dawley rats were divided into four groups: normal group (Normal, n=5), sham-operated group (Sham, n=20), OLT group with 1 hr donor cold ischemic time (n=20), and OLT group with 12 hr donor cold ischemic time (OLTn=20). Expression of mucins and GATA factors in bile ducts was examined by real-time polymerase chain reaction, immunohistochemistry, and immunoblotting. Bile was collected for biochemical analysis, and the histological changes associated with bile duct injury were evaluated. RESULTS In normal bile ducts, Muc1, Muc2, Muc3A, Muc4, and Muc6 mRNA were expressed, whereas Muc5AC mRNA was undetectable. The expression of Muc1, Muc3A, and Muc4 but not Muc2 and Muc6 at mRNA level in graft bile ducts decreased remarkably early after OLT. The decreased expression of Muc1 and Muc4 was further confirmed at protein level by immunohistochemistry and immunoblotting. Downregulation of Muc1 and Muc3A expression by prolonged cold ischemic time was significantly associated with the injury severity scores of large but not small bile ducts. Among six GATA factors, GATA3, GATA4, and GATA6 mRNA were expressed in normal bile ducts. GATA4 and GATA6 mRNA levels decreased significantly after OLT. CONCLUSION Downregulation of Muc1 and Muc3A expression by prolonged cold ischemic time may play a potential role in large bile duct injury early after OLT.
- Published
- 2011
12. Role of Cholangiocyte Bile Acid Transporters in Large Bile Duct Injury After Rat Liver Transplantation
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Dajiang Li, Feng Tian, Shuguang Wang, Geng Chen, Lijin Zhao, Xiaowu Li, Long Cheng, and Zipei Liu
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Male ,medicine.medical_specialty ,Organic anion transporter 1 ,medicine.drug_class ,medicine.medical_treatment ,Organic Anion Transporters, Sodium-Dependent ,Apoptosis ,Liver transplantation ,Polymerase Chain Reaction ,Gastroenterology ,Cholangiocyte ,Bile Acids and Salts ,Rats, Sprague-Dawley ,Pathogenesis ,Internal medicine ,medicine ,Animals ,Bile ,DNA Primers ,Transplantation ,Symporters ,biology ,Bile acid ,Bile duct ,Organ Preservation ,Immunohistochemistry ,Liver Transplantation ,Rats ,Bile Ducts, Intrahepatic ,medicine.anatomical_structure ,Liver ,Biliary tract ,biology.protein ,RNA - Abstract
BACKGROUND: The pathogenesis of nonanastomotic strictures with a patent hepatic artery remains to be investigated. This study focuses on the role of cholangiocyte bile acid transporters in bile duct injury after liver transplantation. METHODS: Sprague-Dawley rats were divided into three groups (n=20 for each): the sham-operated group (Sham), the transplant group with 1-hr donor liver cold preservation (CP-1h), and the transplant group with 12-hr donor liver cold preservation (CP-12h). Bile was collected for biochemical analysis. The histopathologic evaluation of bile duct injury was performed and the cholangiocyte bile acid transporters apical sodium-dependent bile acid transporter (ASBT), ileal lipid binding protein (ILBP), and Ostalpha/Ostbeta were investigated. RESULTS.: The immunohistochemical assay suggested that ASBT and ILBP were expressed exclusively on large bile duct epithelial cells, whereas Ostalpha and Ostbeta were expressed on both small and large bile ducts. Western blot and quantitative polymerase chain reaction analysis showed that the expression levels of these transporters dramatically decreased after transplantation. It took seven to 14 days for ILBP, Ostalpha, and Ostbeta to recover, whereas ASBT recovered within 3 days and even reached a peak above the normal level seven days after operation. In the CP-12h group, the ratios of the ASBT/ILBP, ASBT/Ostalpha and ASBT/Ostbeta expression levels were correlated with the injury severity scores of large but not small bile ducts. CONCLUSIONS: The results suggest that the unparallel alteration of cholangiocyte bile acid transporters may play a potential role in large bile duct injury after liver transplantation with prolonged donor liver preservation.
- Published
- 2010
13. LPS Increases MUC5AC by TACE/TGF-α/EGFR Pathway in Human Intrahepatic Biliary Epithelial Cell
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Peng Jiang, Yu He, Shuguang Wang, Hong Chang, Feng Tian, Xiaobin Feng, Jianwei Li, Zipei Liu, Xin Zhao, and Fei Guo
- Subjects
Lipopolysaccharides ,medicine.medical_specialty ,TGF alpha ,Article Subject ,lcsh:Medicine ,Biology ,ADAM17 Protein ,Lithiasis ,Mucin 5AC ,digestive system ,General Biochemistry, Genetics and Molecular Biology ,Growth factor receptor ,Western blot ,Internal medicine ,medicine ,Humans ,Cells, Cultured ,General Immunology and Microbiology ,medicine.diagnostic_test ,Bile duct ,lcsh:R ,Epithelial Cells ,General Medicine ,Transforming Growth Factor alpha ,medicine.disease ,Up-Regulation ,ErbB Receptors ,ADAM Proteins ,Endocrinology ,medicine.anatomical_structure ,Bile Ducts, Intrahepatic ,Gene Expression Regulation ,Cancer research ,Immunohistochemistry ,Tumor necrosis factor alpha ,sense organs ,Hepatolithiasis ,Transforming growth factor ,Signal Transduction ,Research Article - Abstract
Background. Mucin 5AC (MUC5AC) overproduction plays important roles in stone formation and recurrence of hepatolithiasis. We aim to investigate the involved mechanism and the potential target to block this process.Methods. 42 bile duct samples from hepatolithiasis and 15 normal bile duct samples from hemangioma patients were collected for detecting MUC5AC expression by immunohistochemistry. MUC5AC and phosphoepidermal growth factor receptor (pEGFR) expressions in human intrahepatic biliary epithelial cells (HIBECs) cultured with or without lipopolysaccharide (LPS) were detected by real-time PCR and western blot analysis. Transforming growth factor-α(TGF-α) secretion in HIBECs was detected by ELISA.Results. MUC5AC was overexpressed in bile ducts of hepatolithiasis samples compared with bile ducts from hemangioma samples. LPS upregulated MUC5AC expression in HIBECs. LPS promoted EGFR activation, and inhibiting EGFR activation by AG1478 significantly decreased LPS-induced MUC5AC overexpression in HIBECs. Moreover, LPS increased TGF-αsecretion, and inhibiting tumor necrosis factor-αconverting enzyme (TACE), which has been implicated in ectodomain cleavage of TGF-α, significantly inhibited LPS-induced EGFR activation and subsequent MUC5AC overexpression in HIBECs.Conclusion. Our results suggested that LPS increases MUC5AC expression through the TACE/TGF-α/EGFR pathway in HIBECs. This new finding might give light to the prevention of stone formation and recurrence of hepatolithiasis.
- Published
- 2013
14. Surgical treatment of congenital biliary duct cyst
- Author
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Zhihua Li, Shuguang Wang, De-chun Wang, Dajiang Li, Ping Bie, Zipei Liu, Jian Chen, Shuguo Zheng, and Yu He
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Adult ,Male ,medicine.medical_specialty ,Time Factors ,Adolescent ,Blood Loss, Surgical ,Young Adult ,Cholelithiasis ,Internal medicine ,medicine ,Humans ,Cyst ,Choledochal cysts ,lcsh:RC799-869 ,Laparoscopic resection ,Young adult ,Child ,Laparoscopy ,Aged ,Retrospective Studies ,Aged, 80 and over ,Chi-Square Distribution ,Congenital biliary duct cyst ,medicine.diagnostic_test ,business.industry ,General surgery ,Total cyst excision ,Age Factors ,Gastroenterology ,Infant ,Retrospective cohort study ,General Medicine ,Middle Aged ,Hepatology ,medicine.disease ,Surgery ,Treatment Outcome ,Child, Preschool ,Choledochal Cyst ,lcsh:Diseases of the digestive system. Gastroenterology ,Female ,business ,Chi-squared distribution ,Research Article - Abstract
Background It is acknowledged that total cyst excision is a safe and ideal surgical treatment for congenital biliary duct cyst, compared to simple internal drainage. The aim of this study was to determine the optimal operation occasion and the effect of laparoscopy on congenital biliary duct cyst based upon total cyst excision. Methods From January 2002 to January 2011, 217 patients were admitted to Southwest Hospital for congenital biliary duct cyst. To determine the optimal surgery occasion, we divided these subjects into three groups, the infant group (age ≤ 3 years), the immaturity group (3 < age ≤ 18 years), and the maturity group (age > 18 years), and then evaluated the feasibility, risk and long-term outcome after surgery in the three groups. To analyze the effect of laparoscopic technique on congenital biliary duct cyst, we divided the patients into the laparoscopy and the open surgery groups. Results Among the three groups, the morbidity from cholangiolithiasis before surgical treatment had obvious discrepancy (p < 0.05) (lowest in the infant group), and intraoperative blood loss also had apparent diversity (p < 0.05). Furthermore, long-term outcomes (secondary cholangiolithiasis, stoma stenosis and cholangiocarcinoma) showed no significant difference between different groups (p > 0.05). Similarly, no significant discrepancy was observed in the morbidity from postoperative complications or long-term postoperative complications (p > 0.05) between the laparoscopic and the open surgery groups. Conclusions We conclude that total cyst excision should be performed as early as possible. The optimal treatment occasion is the infant period, and laparoscopic resection may be a new safe and feasible minimally invasive surgery for this disease.
- Published
- 2012
15. Epithelial-mesenchymal transition induced by hepatitis C virus core protein in cholangiocarcinoma
- Author
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Dajiang Li, Shuguang Wang, Ying Hong Gao, Wei-wei Jiang, Zipei Liu, Long Cheng, Hongye Wu, Tianyu Li, Zhan-feng Gao, Lijin Zhao, and Feng Tian
- Subjects
Male ,Blotting, Western ,Biology ,Adenocarcinoma ,Virus ,Epithelium ,Cholangiocarcinoma ,Immunoenzyme Techniques ,Mesoderm ,RNA interference ,Cell Movement ,Cell Adhesion ,Tumor Cells, Cultured ,Gene silencing ,Humans ,Epithelial–mesenchymal transition ,RNA, Messenger ,RNA, Small Interfering ,LOXL2 ,Reverse Transcriptase Polymerase Chain Reaction ,Viral Core Proteins ,Mesenchymal stem cell ,Middle Aged ,Molecular biology ,Blot ,Bile Ducts, Intrahepatic ,Oncology ,Bile Duct Neoplasms ,Cell culture ,Lymphatic Metastasis ,Surgery ,Female ,Amino Acid Oxidoreductases ,Hepatitis C Antigens - Abstract
Cholangiocarcinoma (CC) is associated with chronic hepatitis C virus (HCV) infection. We investigated the effect of hepatitis C virus core protein (HCVc) on epithelial-mesenchymal transition (EMT) in CC and tried to identify its target trigger. First, we examined expression of HCVc and epithelial and mesenchymal markers in CC tissues. Then we transient-transfected HCVc gene into a CC cell line and examined expression of lysyl oxidase-like 2 (LOXL2) and epithelial and mesenchymal markers by quantitative real-time polymerase chain reaction (PCR) and Western blotting. Finally, LOXL2 gene silencing was shown in QBC939/HCVc cells by RNA interference (RNAi), and we further examined expression of epithelial and mesenchymal markers by quantitative real-time PCR and Western blotting. Through immunohistochemical staining, the present study showed that HCVc is significantly associated with CC invasion and metastasis. In vitro study showed that HCVc expression induces EMT in CC cell line QBC939, and a mechanism through LOXL2 pathway is suggested. Expression of HCVc was significantly correlated with greater migratory and invasive potential of CC cells. These observations indicate that HCVc plays a critical role in promoting invasion and metastasis of CC cells.
- Published
- 2009
16. 67 laminin receptor promotes the malignant potential of tumour cells up-regulating lysyl oxidase-like 2 expression in cholangiocarcinoma.
- Author
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Jing Xu, Dajing Li, Xiaowu Li, Zipei Liu, Tianyu Li, Peng Jiang, Qiang He, Feng Tian, Yang Gao, Dechun Wang, and Shuguang Wang
- Abstract
Background 67 laminin receptor (67LR) plays an important role in the invasion and metastasis of cholangiocarcinoma, but its mechanism remains unclear. Aims We investigated the clinical significance of 67LR and its relation to lysyl oxidase-like 2 (LOXL2) in 67LR-mediated invasion and metastasis in cholangiocarcinoma. Methods The clinical significance of 67LR and LOXL2 expression and the prognosis of patients were investigated in 73 cancerous and 32 paracancerous tissues by immunohistochemistry. The impact of LOXL2 on invasion, metastasis and 67LR expression was evaluated in cholangiocarcinoma cells by shRNA or expressed-plasmid transfection. Results Expression of 67LR was recognized in 35.62% cholangiocarcinoma tissue, and none in paracancerous tissues. LOXL2 was positively correlated with expression of 67LR. Expression of 67LR or LOXL2 in cholangiocarcinomas was significantly associated with lymph node metastasis, differentiation and poor overall survival. Cox analysis showed that 67LR can act as an independent prognostic biomarker of prognosis in cholangiocarcinoma patients. Expression of LOXL2 decreased by knockdown of 67LR and increased by overexpression of 67LR in cholangiocarcinoma cells. Knockdown of LOXL2 reduced invasion and metastasis in vitro and in vivo. Conclusion 67LR may regulate the expression of LOXL2 to promote invasion and metastasis in cholangiocarcinoma cells. It could be used as an independent prognostic marker in cholangiocarcinoma patients. [ABSTRACT FROM AUTHOR]
- Published
- 2014
- Full Text
- View/download PDF
17. LPS Increases MUC5AC by TACE/TGF-α/EGFR Pathway in Human Intrahepatic Biliary Epithelial Cell.
- Author
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Zipei Liu, Feng Tian, Xiaobin Feng, Yu He, Peng Jiang, Jianwei Li, Fei Guo, Xin Zhao, Hong Chang, and Shuguang Wang
- Abstract
Background. Mucin 5AC (MUC5AC) overproduction plays important roles in stone formation and recurrence of hepatolithiasis. We aim to investigate the involved mechanism and the potential target to block this process. Methods. 42 bile duct samples from hepatolithiasis and 15 normal bile duct samples from hemangioma patients were collected for detecting MUC5AC expression by immunohistochemistry. MUC5AC and phosphoepidermal growth factor receptor (pEGFR) expressions in human intrahepatic biliary epithelial cells (HIBECs) cultured with or without lipopolysaccharide (LPS) were detected by real-time PCR and western blot analysis. Transforming growth factor-α (TGF-α) secretion in HIBECs was detected by ELISA. Results. MUC5AC was overexpressed in bile ducts of hepatolithiasis samples compared with bile ducts from hemangioma samples. LPS upregulated MUC5AC expression in HIBECs. LPS promoted EGFR activation, and inhibiting EGFR activation by AG1478 significantly decreased LPS-induced MUC5AC overexpression in HIBECs. Moreover, LPS increased TGF-α secretion, and inhibiting tumor necrosis factor-α converting enzyme (TACE), which has been implicated in ectodomain cleavage of TGF-α, significantly inhibited LPS-induced EGFR activation and subsequent MUC5AC overexpression in HIBECs. Conclusion. Our results suggested that LPS increases MUC5AC expression through the TACE/TGF-α/EGFR pathway in HIBECs. This new finding might give light to the prevention of stone formation and recurrence of hepatolithiasis. [ABSTRACT FROM AUTHOR]
- Published
- 2013
- Full Text
- View/download PDF
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