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Your search keyword '"Zhu, Wen-Kai"' showing total 25 results
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25 results on '"Zhu, Wen-Kai"'

5. Basic Model Design of Online Monitoring System for Mixed Radiation Field

Author

Cheng, Jin-Xing, Zhu, Wen-Kai, Wang, Qing-Bo, Wen, Wei-Wei, Wu, You-Peng, Yu, Ai, Angrisani, Leopoldo, Series Editor, Arteaga, Marco, Series Editor, Panigrahi, Bijaya Ketan, Series Editor, Chakraborty, Samarjit, Series Editor, Chen, Jiming, Series Editor, Chen, Shanben, Series Editor, Chen, Tan Kay, Series Editor, Dillmann, Rüdiger, Series Editor, Duan, Haibin, Series Editor, Ferrari, Gianluigi, Series Editor, Ferre, Manuel, Series Editor, Hirche, Sandra, Series Editor, Jabbari, Faryar, Series Editor, Jia, Limin, Series Editor, Kacprzyk, Janusz, Series Editor, Khamis, Alaa, Series Editor, Kroeger, Torsten, Series Editor, Li, Yong, Series Editor, Liang, Qilian, Series Editor, Martín, Ferran, Series Editor, Ming, Tan Cher, Series Editor, Minker, Wolfgang, Series Editor, Misra, Pradeep, Series Editor, Möller, Sebastian, Series Editor, Mukhopadhyay, Subhas, Series Editor, Ning, Cun-Zheng, Series Editor, Nishida, Toyoaki, Series Editor, Pascucci, Federica, Series Editor, Qin, Yong, Series Editor, Seng, Gan Woon, Series Editor, Speidel, Joachim, Series Editor, Veiga, Germano, Series Editor, Wu, Haitao, Series Editor, Zamboni, Walter, Series Editor, Zhang, Junjie James, Series Editor, Xu, Yang, editor, Sun, Yongbin, editor, Liu, Yanyang, editor, Gao, Feng, editor, Gu, Pengfei, editor, and Liu, Zheming, editor

Published
2021
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6. Multi-ray Intelligent Monitoring System for Mixed Radiation Field Measurements

Author

Cheng, Jin-Xing, Wu, You-Peng, Wang, Qing-Bo, Wen, Wei-Wei, Yu, Ai, Qiu, Rui, Zhang, Yue, Zhu, Wen-Kai, Angrisani, Leopoldo, Series Editor, Arteaga, Marco, Series Editor, Panigrahi, Bijaya Ketan, Series Editor, Chakraborty, Samarjit, Series Editor, Chen, Jiming, Series Editor, Chen, Shanben, Series Editor, Chen, Tan Kay, Series Editor, Dillmann, Rüdiger, Series Editor, Duan, Haibin, Series Editor, Ferrari, Gianluigi, Series Editor, Ferre, Manuel, Series Editor, Hirche, Sandra, Series Editor, Jabbari, Faryar, Series Editor, Jia, Limin, Series Editor, Kacprzyk, Janusz, Series Editor, Khamis, Alaa, Series Editor, Kroeger, Torsten, Series Editor, Li, Yong, Series Editor, Liang, Qilian, Series Editor, Martín, Ferran, Series Editor, Ming, Tan Cher, Series Editor, Minker, Wolfgang, Series Editor, Misra, Pradeep, Series Editor, Möller, Sebastian, Series Editor, Mukhopadhyay, Subhas, Series Editor, Ning, Cun-Zheng, Series Editor, Nishida, Toyoaki, Series Editor, Pascucci, Federica, Series Editor, Qin, Yong, Series Editor, Seng, Gan Woon, Series Editor, Speidel, Joachim, Series Editor, Veiga, Germano, Series Editor, Wu, Haitao, Series Editor, Zamboni, Walter, Series Editor, Zhang, Junjie James, Series Editor, Xu, Yang, editor, Sun, Yongbin, editor, Liu, Yanyang, editor, Gao, Feng, editor, Gu, Pengfei, editor, and Liu, Zheming, editor

Published
2021
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13. Hexokinase 3 Dysfunction Promotes Tumorigenesis and Immune Escape by Regulating Monocyte/Macrophage Infiltration of Clear Cell Renal Cell Carcinoma Microenvironment

Author

Xu, Wen-Hao, primary, Liu, WangRui, additional, Xu, Yue, additional, Tian, Xi, additional, Anwaier, Aihetaimujiang, additional, Su, Jia-Qi, additional, Zhu, Wen-Kai, additional, Shi, Guo-Hai, additional, Zhong, Chunlong, additional, Qu, Yuan-Yuan, additional, Zhang, Hai-Liang, additional, and Ye, Ding-Wei, additional

Published
2021
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14. Hexokinase 3 dysfunction promotes tumorigenesis and immune escape by upregulating monocyte/macrophage infiltration into the clear cell renal cell carcinoma microenvironment

Author

Xu, Wenhao, primary, Liu, Wang-Rui, additional, Xu, Yue, additional, Tian, Xi, additional, Anwaier, Aihetaimujiang, additional, Su, Jia-Qi, additional, Zhu, Wen-Kai, additional, Shi, Guo-Hai, additional, Wei, Gao-Meng, additional, Huang, Yong-Ping, additional, Qu, Yuan-Yuan, additional, Zhang, Hai-Liang, additional, and Ye, Ding-Wei, additional

Published
2021
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15. Large‐scale transcriptome profiles reveal robust 20‐signatures metabolic prediction models and novel role of G6PC in clear cell renal cell carcinoma

Author

Xu, Wen‐Hao, primary, Xu, Yue, additional, Tian, Xi, additional, Anwaier, Aihetaimujiang, additional, Liu, Wang‐Rui, additional, Wang, Jun, additional, Zhu, Wen‐Kai, additional, Cao, Da‐Long, additional, Wang, Hong‐Kai, additional, Shi, Guo‐Hai, additional, Qu, Yuan‐Yuan, additional, Zhang, Hai‐Liang, additional, and Ye, Ding‐Wei, additional

Published
2020
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16. Fatty acid synthase (FASN), a novel signature for visceral adipose tissues, regulates proliferation, migration and predicts prognosis of clear cell renal cell carcinoma

Author

Xu, Wen-Hao, primary, Anwaier, Aihetaimujiang, additional, Wang, Hong-Kai, additional, Wang, Jun, additional, Zhu, Wen-Kai, additional, Tian, Xi, additional, Ma, Chung-Guang, additional, Wan, Fang-Ning, additional, Cao, Da-Long, additional, Shi, Guo-Hai, additional, Qu, Yuan-Yuan, additional, Zhang, Hai-Liang, additional, and Ye, Ding-Wei, additional

Published
2020
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22. Investigation of the structural phase transition of MnBi and MnBiAl films

Author

Xu Yao, Zhang Xiao-wei, Zhu Wen-kai, Wang Yin-jun, and Yang Hui-sheng

Subjects
Condensed Matter::Materials Science, Structural phase, Phase transition, Materials science, chemistry, Condensed matter physics, Aluminium, Phase (matter), General Physics and Astronomy, chemistry.chemical_element, Atmospheric temperature range
Abstract

Structural phase transition of MnBi and MnBiAl films has been investigated in the temperature range 30-400°C. The transition from low-temperature phase to high-temperature phase occurs in both the films, but the phase transitions appear at different temperatures. According to the measurement of Kerr rotation as a function of thermal annealing and the phase transition process, it is shown that the transition is irreversible for MnBi film and reversible for MnBiAl film under our experimental condition. For this reason, we speculate that the aluminum added in MnBi makes the bonding force of Mn atoms with their neighbors stronger, which may be the cause for the difference of the phase transition between MnBi and MnBiAl films.

Published
1995

25. CircHIPK3 promotes neuroinflammation through regulation of the miR-124-3p/STAT3/NLRP3 signaling pathway in Parkinson's disease.

Author

Zhang YJ, Zhu WK, Qi FY, and Che FY

Subjects
Humans, NLR Family, Pyrin Domain-Containing 3 Protein genetics, NLR Family, Pyrin Domain-Containing 3 Protein metabolism, Neuroinflammatory Diseases, Interleukin-6 metabolism, STAT3 Transcription Factor metabolism, Tumor Necrosis Factor-alpha metabolism, Lipopolysaccharides, In Situ Hybridization, Fluorescence, Signal Transduction, Caspase 1 metabolism, Parkinson Disease genetics, Neurodegenerative Diseases, Neuroblastoma, MicroRNAs genetics, MicroRNAs metabolism
Abstract

Background: Parkinson's disease (PD) is characterized as a neurodegenerative disease; however, the mechanisms regarding its pathogenesis have not been fully explored., Objectives: To explore the role of circular RNA homeodomain interacting protein kinase 3 (circHIPK3) in the progression of PD., Material and Methods: The circHIPK3 and microRNA-124 (miR-124) expression in human serum and cerebral fluid was detected using real-time quantitative reverse transcription polymerase chain reaction (qRT-PCR) in 92 PD patients and 95 controls. The circHIPK3 was overexpressed and/or silenced in cells to explore its molecular mechanisms and effects on neuroinflammation. The production of intracellular reactive oxygen species (ROS) was assessed using 2',7'-dichlorodihydrofluorescein diacetate (DCFH-DA) staining. Interleukin 6 (IL-6), IL-1β and tumor necrosis factor alpha (TNF-α) production in BV2 cells after the indicated treatment was measured using enzyme-linked immunosorbent assay (ELISA). The protein expression of microglia markers (cluster of differentiation molecule 11b (CD11b) and ionized calcium-binding adapter molecule 1 (Iba-1)), pyroptosis-related factors, NLR family pyrin domain containing 3 (NLRP3), apoptosis-associated speck-like protein containing C-terminal caspase recruitment domain (ASC), and caspase-1, signal transducer and activator of transcription 3 (STAT3), and phosphorylated STAT3 (p-STAT3) were examined using western blot analysis. Furthermore, the interaction between circHIPK3, miR-124 and STAT3 was predicted with bioinformatics and examined using fluorescence in situ hybridization (FISH), luciferase reporter assays, RNA pull-down, and RNA immunoprecipitation (RIP)., Results: The expression of circHIPK3 in human serum and cerebral fluids was significantly higher than in controls, whereas miR-124 expression was drastically reduced. In addition, lipopolysaccharide (LPS)-treated BV2 cells exhibited higher expression of circHIPK3 and lower miR-124 expression. The SH-SY5Y cells exhibited a significantly impaired viability and elevated apoptotic rate, along with an upregulation of circHIPK3 and a downregulation of miR-124 expression after being treated with supernatants collected from LPS-treated BV2 cells. The upregulation of circHIPK3 increased IL-6, IL-1β and TNF-α secretion in BV2 cells. The protein expressions of microglia markers (CD11b and Iba-1), as well as pyroptosis-related factors, NLRP3, caspase-1, and ASC, were also increased following the expression of circHIPK3. All these effects were reversed by the addition of miR-124., Conclusions: The circHIPK3 enhances neuroinflammation by sponging miR-124 and regulating the miR-124-mediated STAT3/NALP3 pathway in PD.

Published
2023
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