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46 results on '"Zhang, Yunyuan"'

7. [Corrigendum] S100A9 promotes the proliferation and invasion of HepG2 hepatocellular carcinoma cells via the activation of the MAPK signaling pathway

Author

Wu, Rui, primary, Duan, Liang, additional, Ye, Liwei, additional, Wang, Haiyang, additional, Yang, Xia, additional, Zhang, Yunyuan, additional, Chen, Xian, additional, Zhang, Yan, additional, Weng, Yaguang, additional, Luo, Jingyong, additional, Tang, Min, additional, Shi, Qiong, additional, He, Tongchuan, additional, and Zhou, Lan, additional

Published
2023
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9. P‐11.3: Splicing Seam Simulation Analysis of Glass‐based Micro LED Splicing Screen.

Author

zhang, Yunyuan, Zhou, Wenqi, Qian, Haochen, Liang, Zhixuan, Chen, Zemin, Huo, Sitao, and Wu, Tianyi

Subjects
LED displays
Abstract

This paper mainly studied the splicing seam structure of the splicing screen, analyzed the key factors of the invisible splicing seam: the thickness of the transparent encapsulated layer, the slope length of the edge of the transparent encapsulated layer, and the length of the side encapsulated layer covering the transparent encapsulated layer. Optical simulations were built to analyze the influence of three factors on the visible degree of splicing seam. The simulation results can provide references for the actual production of splicing Micro LED display products. [ABSTRACT FROM AUTHOR]

Published
2024
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10. A Preprocessing Method of EEG Based on EMD-ICA in BCI

Author

Yang, Banghua, He, Liangfei, Wang, Qian, Song, Chunting, Zhang, Yunyuan, Junqueira Barbosa, Simone Diniz, Series editor, Chen, Phoebe, Series editor, Cuzzocrea, Alfredo, Series editor, Du, Xiaoyong, Series editor, Filipe, Joaquim, Series editor, Kara, Orhun, Series editor, Kotenko, Igor, Series editor, Sivalingam, Krishna M., Series editor, Ślęzak, Dominik, Series editor, Washio, Takashi, Series editor, Yang, Xiaokang, Series editor, Ma, Shiwei, editor, Jia, Li, editor, Li, Xin, editor, Wang, Ling, editor, Zhou, Huiyu, editor, and Sun, Xin, editor

Published
2014
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33. Inactivation of the Phosphatidylinositol 3‐Kinase/Akt Pathway is Involved in BMP9‐mediated Tumor‐suppressive Effects in Gastric Cancer Cells

Author

Duan, Liang, primary, Ye, Liwei, additional, Wu, Rui, additional, Wang, Haiyan, additional, Li, Xueru, additional, Li, Huan, additional, Yuan, Shimei, additional, Zha, He, additional, Sun, Hui, additional, Zhang, Yunyuan, additional, Chen, Xian, additional, Zhang, Yan, additional, and Zhou, Lan, additional

Published
2015
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35. Bone morphogenetic protein 2 inhibits the proliferation and growth of human colorectal cancer cells

Author

ZHANG, YUNYUAN, primary, CHEN, XIAN, additional, QIAO, MIN, additional, ZHANG, BING-QIANG, additional, WANG, NING, additional, ZHANG, ZHONGLIN, additional, LIAO, ZHAN, additional, ZENG, LIYI, additional, DENG, YOULIN, additional, DENG, FANG, additional, ZHANG, JUNHUI, additional, YIN, LIANGJUN, additional, LIU, WEI, additional, ZHANG, QIAN, additional, YAN, ZHENGJIAN, additional, YE, JIXING, additional, WANG, ZHONGLIANG, additional, ZHOU, LAN, additional, LUU, HUE H., additional, HAYDON, REX C., additional, HE, TONG-CHUAN, additional, and ZHANG, HONGYU, additional

Published
2014
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38. Correction: S100A8 and S100A9 Are Associated with Colorectal Carcinoma Progression and Contribute to Colorectal Carcinoma Cell Survival and Migration via Wnt/β-Catenin Pathway

Author

Duan, Liang, primary, Wu, Rui, additional, Ye, Liwei, additional, Wang, Haiyan, additional, Yang, Xia, additional, Zhang, Yunyuan, additional, Chen, Xian, additional, Zuo, Guowei, additional, Zhang, Yan, additional, Weng, Yaguang, additional, Luo, Jinyong, additional, Tang, Min, additional, Shi, Qiong, additional, He, Tongchuan, additional, and Zhou, Lan, additional

Published
2013
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39. S100A8 and S100A9 Are Associated with Colorectal Carcinoma Progression and Contribute to Colorectal Carcinoma Cell Survival and Migration via Wnt/β-Catenin Pathway

Author

Duan, Liang, primary, Wu, Rui, additional, Ye, Liwei, additional, Wang, Haiyan, additional, Yang, Xia, additional, Zhang, Yunyuan, additional, Chen, Xian, additional, Zuo, Guowei, additional, Zhang, Yan, additional, Weng, Yaguang, additional, Luo, Jinyong, additional, Tang, Min, additional, Shi, Qiong, additional, He, Tongchuan, additional, and Zhou, Lan, additional

Published
2013
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40. S100A9 promotes the proliferation and invasion of HepG2 hepatocellular carcinoma cells via the activation of the MAPK signaling pathway

Author

WU, RUI, primary, DUAN, LIANG, additional, YE, LIWEI, additional, WANG, HAIYANG, additional, YANG, XIA, additional, ZHANG, YUNYUAN, additional, CHEN, XIAN, additional, ZHANG, YAN, additional, WENG, YAGUANG, additional, LUO, JINGYONG, additional, TANG, MIN, additional, SHI, QIONG, additional, HE, TONGCHUAN, additional, and ZHOU, LAN, additional

Published
2013
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43. High Prevalence of Plasmid-Mediated Quinolone Resistance Determinants qnr , aac(6 ′ ) - Ib-cr , and qepA among Ceftiofur-Resistant Enterobacteriaceae Isolates from Companion and Food-Producing Animals

Author

Ma, Junying, primary, Zeng, Zhenling, additional, Chen, Zhangliu, additional, Xu, Xiaogang, additional, Wang, Xiaoying, additional, Deng, Yuting, additional, Lü, Dianhong, additional, Huang, Liangzong, additional, Zhang, Yunyuan, additional, Liu, Jianhua, additional, and Wang, Minggui, additional

Published
2009
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44. High Prevalence of Plasmid-Mediated Quinolone Resistance Determinants qnr, aac(6′)-Ib-cr, and qepAamong Ceftiofur-Resistant EnterobacteriaceaeIsolates from Companion and Food-Producing Animals

Author

Ma, Junying, Zeng, Zhenling, Chen, Zhangliu, Xu, Xiaogang, Wang, Xiaoying, Deng, Yuting, Lü, Dianhong, Huang, Liangzong, Zhang, Yunyuan, Liu, Jianhua, and Wang, Minggui

Abstract

ABSTRACTThree kinds of plasmid-mediated quinolone resistance (PMQR) determinants have been discovered and have been shown to be widely distributed among clinical isolates: qnrgenes, aac(6′)-Ib-cr, and qepA. Few data on the prevalence of these determinants in strains from animals are available. The presence of PMQR genes in isolates from animals was determined by PCR amplification and DNA sequencing. The production of extended-spectrum β-lactamases (ESBLs) and AmpC β-lactamases in the strains was detected, and their genotypes were determined. The genetic environment of PMQR determinants in selected plasmids was analyzed. All samples of ceftiofur-resistant (MICs ≥ 8 μg/ml) isolates of the family Enterobacteriaceaewere selected from 36 companion animals and 65 food-producing animals in Guangdong Province, China, between November 2003 and April 2007, including 89 Escherichia coliisolates, 9 Klebsiella pneumoniaeisolates, and isolates of three other genera. A total of 68.3% (69/101) of the isolates produced ESBLs and/or AmpC β-lactamases, mainly those of the CTX-M and CMY types. Of the 101 strains, PMQR determinants were present in 35 (34.7%) isolates, with qnr, aac(6′)-Ib-cr, and qepAdetected alone or in combination in 8 (7.9%), 19 (18.8%), and 16 (15.8%) strains, respectively. The qnrgenes detected included one qnrB4gene, four qnrB6genes, and three qnrS1genes. Five strains were positive for both aac(6′)-Ib-crand qepA, while one strain was positive for qnrS1, aac(6′)-Ib-cr, and qepA. qnrB6was flanked by two copies of ISCR1with an intervening dfrgene downstream and sul1and qacEΔ1genes upstream. In another plasmid, aac(6′)-Ib-crfollowed intI1and arr-3was downstream. PMQR determinants are highly prevalent in ceftiofur-resistant Enterobacteriaceaestrains isolated from animals in China. This is the first report of the occurrence of PMQR determinants among isolates from companion animals.

Published
2009
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46. S100A6 stimulates proliferation and migration of colorectal carcinoma cells through activation of the MAPK pathways.

Author

Duan L, Wu R, Zou Z, Wang H, Ye L, Li H, Yuan S, Li X, Zha H, Sun H, Zhang Y, Chen X, and Zhou L

Subjects
Animals, Cell Cycle Proteins genetics, Cell Line, Tumor, Cell Movement genetics, Colorectal Neoplasms metabolism, Colorectal Neoplasms pathology, Gene Expression Regulation, Neoplastic, HCT116 Cells, Humans, MAP Kinase Signaling System genetics, Mice, Mitogen-Activated Protein Kinase Kinases genetics, RNA Interference, S100 Calcium Binding Protein A6, S100 Proteins genetics, Cell Cycle Proteins biosynthesis, Cell Proliferation, Colorectal Neoplasms genetics, Mitogen-Activated Protein Kinase Kinases biosynthesis, S100 Proteins biosynthesis
Abstract

The S100A6 protein, a member of the S100 protein family, is overexpressed in many tumors including colorectal carcinoma (CRC). Although recent studies showed that the elevated expression of S100A6 was associated with the stage and lymphatic permeation of CRC, little is known about whether and how S100A6 contributes to CRC development. Here we investigated the S100A6 expression in CRC tissues and cell lines, and explored the molecular mechanisms underlying the role of S100A6 in CRC development by examining cell proliferation and migration in vitro, and tumorigenicity in nude mice. The results show that S100A6 expression was markedly increased in CRC tissues and cell lines compared to normal colon tissues and a normal colon mucosal epithelial cell line, respectively. Recombinant adenovirus-mediated overexpression of S100A6 or treatment with recombinant S100A6 protein in HCT116, a CRC cell line with relative low S100A6 expression, resulted in enhanced cell proliferation and migration, and the mitogen-activated protein kinase (MAPK) activation in vitro, and tumor growth in vivo. Conversely, RNAi-mediated knockdown of S100A6 in LoVo, a CRC cell line with relative high S100A6 expression, resulted in reduced cell proliferation, migration and MAPK activity. S100A6-induced proliferation was partially attenuated by an ERK inhibitor while migration was suppressed by a p38 inhibitor. Taken together, our results suggest that the cellular effects of S100A6 are mediated by the ERK and p38 MAPK pathways, and modulation of these pathways may be employed for CRC prevention and therapy.

Published
2014
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