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2. Corrigendum

3. Large-scale Gender/Age Prediction of Tumblr Users

4. Abundant dynamics of group velocity locked vector solitons from Er-doped fiber laser based on GO/PVA film

5. Correction: MNK1/NODAL Signaling Promotes Invasive Progression of Breast Ductal Carcinoma In Situ

6. MNK1/2 inhibition limits oncogenicity and metastasis of KIT-mutant melanoma

7. Supplementary Data from MNK1/NODAL Signaling Promotes Invasive Progression of Breast Ductal Carcinoma In Situ

8. Data from MNK1/NODAL Signaling Promotes Invasive Progression of Breast Ductal Carcinoma In Situ

10. Impact of the model of long‐term follow‐up care on adherence to guideline‐recommended surveillance among survivors of adolescent and young adult cancers

11. Mutant-selective degradation by BRAF-targeting PROTACs

12. RAF dimer inhibition enhances the antitumor activity of MEK inhibitors in K‐RAS mutant tumors

16. Efficacy and Safety of Tranexamic Acid in Reducing Blood Loss of Lower Extremity Osteotomy in Peri‐acetabulum and High Tibia: A Systematic Review and Meta‐analysis

17. Clinical study on hematoma puncture and catheter drainage in treatment of intracerebral hemorrhage under CT real-time guide

18. A population-based study of the risk of osteoporosis and fracture with dutasteride and finasteride

20. Abundant dynamics of group velocity locked vector solitons from Er-doped fiber laser based on GO/PVA film

21. Back Frame Optimization of a Large Radio Telescope Based on Force Cone Method

23. Supplementary Table S2 from Gain-of-Function Genetic Alterations of G9a Drive Oncogenesis

24. Supplementary Figure S1-S10 from Gain-of-Function Genetic Alterations of G9a Drive Oncogenesis

25. Data from Gain-of-Function Genetic Alterations of G9a Drive Oncogenesis

26. Data from MNK1/NODAL Signaling Promotes Invasive Progression of Breast Ductal Carcinoma In Situ

27. Supplementary Data from MNK1/NODAL Signaling Promotes Invasive Progression of Breast Ductal Carcinoma In Situ

28. Rare BRAF mutations in pancreatic neuroendocrine tumors may predict response to RAF and MEK inhibition.

29. Prediction of Chemical Composition in Tea Based on Image Processing Technology

30. Supplementary Methods, Figures 1 - 5, Tables 1 - 5 from Diverse and Targetable Kinase Alterations Drive Histiocytic Neoplasms

31. Data from Rapid Induction of Apoptosis by PI3K Inhibitors Is Dependent upon Their Transient Inhibition of RAS–ERK Signaling

32. Supplementary Material from A Secondary Mutation in BRAF Confers Resistance to RAF Inhibition in a BRAFV600E-Mutant Brain Tumor

33. Supplementary Materials and Methods from Rapid Induction of Apoptosis by PI3K Inhibitors Is Dependent upon Their Transient Inhibition of RAS–ERK Signaling

34. Supplementary Figure S6 from Rapid Induction of Apoptosis by PI3K Inhibitors Is Dependent upon Their Transient Inhibition of RAS–ERK Signaling

36. Data from Allele-Specific Mechanisms of Activation of MEK1 Mutants Determine Their Properties

37. Supplementary Figure Legends from Rapid Induction of Apoptosis by PI3K Inhibitors Is Dependent upon Their Transient Inhibition of RAS–ERK Signaling

40. Data from V211D Mutation in MEK1 Causes Resistance to MEK Inhibitors in Colon Cancer

42. Supplementary Data from Leveraging Systematic Functional Analysis to Benchmark an In Silico Framework Distinguishes Driver from Passenger MEK Mutants in Cancer

43. Data from Mechanisms of Acquired Resistance to BRAF V600E Inhibition in Colon Cancers Converge on RAF Dimerization and Are Sensitive to Its Inhibition

44. Supplementary figures from Mechanisms of Acquired Resistance to BRAF V600E Inhibition in Colon Cancers Converge on RAF Dimerization and Are Sensitive to Its Inhibition

45. Data from Leveraging Systematic Functional Analysis to Benchmark an In Silico Framework Distinguishes Driver from Passenger MEK Mutants in Cancer

46. Data from Combined Inhibition of SHP2 and MEK Is Effective in Models of NF1-Deficient Malignant Peripheral Nerve Sheath Tumors

48. Supplementary Figure Legends from Mechanisms of Acquired Resistance to BRAF V600E Inhibition in Colon Cancers Converge on RAF Dimerization and Are Sensitive to Its Inhibition

49. Data Supplement from Loss of NF1 in Cutaneous Melanoma Is Associated with RAS Activation and MEK Dependence

50. Inhibiting the MNK1/2-eIF4E axis impairs melanoma phenotype switching and potentiates antitumor immune responses

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