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196 results on '"Zahm, S H"'

3. Cigarette smoking and risk of Hodgkin lymphoma and its subtypes: a pooled analysis from the International Lymphoma Epidemiology Consortium (InterLymph)

Author

Kamper-Jørgensen, M., Rostgaard, K., Glaser, S. L., Zahm, S. H., Cozen, W., Smedby, K. E., Sanjosé, S., Chang, E. T., Zheng, T., La Vecchia, C., Serraino, D., Monnereau, A., Kane, E. V., Miligi, L., Vineis, P., Spinelli, J. J., McLaughlin, J. R., Pahwa, P., Dosman, J. A., Vornanen, M., Foretova, L., Maynadie, M., Staines, A., Becker, N., Nieters, A., Brennan, P., Boffetta, P., Cocco, P., and Hjalgrim, H.

Published
2013
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6. Cigarette smoking and risk of Hodgkin lymphoma and its subtypes:a pooled analysis from the International Lymphoma Epidemiology Consortium (InterLymph)

Author

Kamper-Jørgensen, Mads, Rostgaard, K, Glaser, S L, Zahm, S H, Cozen, W, Smedby, K E, Sanjosé, S, Chang, E T, Zheng, T, La Vecchia, C, Serraino, D, Monnereau, A, Kane, E V, Miligi, L, Vineis, P, Spinelli, J J, McLaughlin, J R, Pahwa, P, Dosman, J A, Vornanen, M, Foretova, L, Maynadie, M, Staines, A, Becker, N, Nieters, A, Brennan, P, Boffetta, P, Cocco, P, Hjalgrim, H, Kamper-Jørgensen, Mads, Rostgaard, K, Glaser, S L, Zahm, S H, Cozen, W, Smedby, K E, Sanjosé, S, Chang, E T, Zheng, T, La Vecchia, C, Serraino, D, Monnereau, A, Kane, E V, Miligi, L, Vineis, P, Spinelli, J J, McLaughlin, J R, Pahwa, P, Dosman, J A, Vornanen, M, Foretova, L, Maynadie, M, Staines, A, Becker, N, Nieters, A, Brennan, P, Boffetta, P, Cocco, P, and Hjalgrim, H

Abstract

The etiology of Hodgkin lymphoma (HL) remains incompletely characterized. Studies of the association between smoking and HL have yielded ambiguous results, possibly due to differences between HL subtypes.

Published
2013

7. Bracken fern consumption and human bladder cancer

Author

Brown, L. M., Zahm, S. H., Hoover, R. N., and Fraumeni, J. F.

Subjects
Spores, Inhalation Exposure, Guinea Pigs, Risk Assessment, United Kingdom, Rats, Plants, Toxic, Cricetinae, Carcinogens, Animals, Humans, Cattle, Letters, Mutagens
Abstract

To assess the risk to human health of the plant bracken (Pteridium sp).An evaluation of studies of human and animal populations exposed to bracken, together with a review of expert reports and advice to the public.Bracken induced disease has been demonstrated in animals in both laboratory and field studies. Depending on the species, diseases in animals associated with the plant have included; cancers of the alimentary and urogenital tract, lung and breast; haematuria; retinal degeneration; and, thiamine deficiency. Potential exposure of human populations is through: food either directly (people in some parts of the world eat bracken as a traditional dish) or indirectly by consuming animals fed on bracken; milk; water; inhalation and ingestion of spores; and insect vectors. Four studies of human populations (two analytical and two observational) failed to assess adequately confounding factors and other sources of bias, so that conclusions about a risk to human health from bracken cannot firmly be drawn. Establishing exposure is also extremely difficult in populations (such as the United Kingdom) where direct consumption of bracken is rare.Bracken is a common plant worldwide. It is toxic to many animal species and to several organ systems. There is no tumour (or other disease) that is pathognomic of exposure in animals, though cancers of the alimentary and urogenital tract seem to be the most commonly associated. It is not possible to extrapolate from animal models to humans. Studies of human populations, do not establish a clear risk of bracken to human health, largely because of methodological problems. Testing the evidence against traditional criteria of causality only fulfils the criterion of biological plausibility. Despite this, current public information implies a serious risk to human health from bracken, and increasing media coverage of the subject is likely to lead to greater public concern. Further epidemiological studies are required.

Published
1999

9. Atrazine and cancer incidence among pesticide applicators in the Agricultural Health Study (1994-2007)

Author

Freeman, L. B., primary, Rusiecki, J., additional, Hoppin, J., additional, Lubin, J., additional, Koutros, S., additional, Andreotti, G., additional, Zahm, S. H., additional, Hines, C., additional, Coble, J., additional, Barone-Adesi, F., additional, Sloan, J., additional, Sandler, D., additional, Blair, A., additional, and Alavanja, M., additional

Published
2011
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10. Impact of pesticide exposure misclassification on estimates of relative risks in the Agricultural Health Study

Author

Blair, A., primary, Thomas, K., additional, Coble, J., additional, Sandler, D. P., additional, Hines, C. J., additional, Lynch, C. F., additional, Knott, C., additional, Purdue, M. P., additional, Zahm, S. H., additional, Alavanja, M. C. R., additional, Dosemeci, M., additional, Kamel, F., additional, Hoppin, J. A., additional, Freeman, L. B., additional, and Lubin, J. H., additional

Published
2011
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14. Personal Use of Hair Dye and the Risk of Certain Subtypes of Non-Hodgkin Lymphoma

Author

Zhang, Y., primary, Sanjose, S. D., additional, Bracci, P. M., additional, Morton, L. M., additional, Wang, R., additional, Brennan, P., additional, Hartge, P., additional, Boffetta, P., additional, Becker, N., additional, Maynadie, M., additional, Foretova, L., additional, Cocco, P., additional, Staines, A., additional, Holford, T., additional, Holly, E. A., additional, Nieters, A., additional, Benavente, Y., additional, Bernstein, L., additional, Zahm, S. H., additional, and Zheng, T., additional

Published
2008
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18. Genetic variants in caspase genes and susceptibility to non-Hodgkin lymphoma

Author

Lan, Q., primary, Zheng, T., additional, Chanock, S., additional, Zhang, Y., additional, Shen, M., additional, Wang, S. S., additional, Berndt, S. I., additional, Zahm, S. H., additional, Holford, T. R., additional, Leaderer, B., additional, Yeager, M., additional, Welch, R., additional, Hosgood, D., additional, Boyle, P., additional, and Rothman, N., additional

Published
2006
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32. The Agricultural Health Study.

Author

Alavanja, M C, primary, Sandler, D P, additional, McMaster, S B, additional, Zahm, S H, additional, McDonnell, C J, additional, Lynch, C F, additional, Pennybacker, M, additional, Rothman, N, additional, Dosemeci, M, additional, Bond, A E, additional, and Blair, A, additional

Published
1996
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45. Agricultural use of organophosphate pesticides and the risk of non-Hodgkin's lymphoma among male farmers (United States).

Author

Waddell, Barry, Zahm, Shelia, Baris, Dalsu, Weisenburger, Dennis, Holmes, Frederick, Burmeister, Leon, Cantor, Kenneth, Blair, Aaron, Waddell, B L, Zahm, S H, Baris, D, Weisenburger, D D, Holmes, F, Burmeister, L F, Cantor, K P, and Blair, A

Abstract

Objective: Data from three population-based case-control studies conducted in Kansas, Nebraska, Iowa, and Minnesota were pooled to evaluate the relationship between the use of organophosphate pesticides and non-Hodgkin's lymphoma (NHL) among white male farmers.Methods: The data set included 748 cases of non-Hodgkin's lymphoma and 2236 population-based controls. Telephone or in-person interviews were utilized to obtain information on the use of pesticides. Odds ratios (OR) adjusted for age, state of residence, and respondent status, as well as other pesticide use where appropriate, were estimated by logistic regression.Results: Use of organophosphate pesticides was associated with a statistically significant 50% increased risk of NHL, but direct interviews showed a significantly lower risk (OR = 1.2) than proxy interviews (OR = 3.0). Among direct interviews the risk of small lymphocytic lymphoma increased with diazinon use (OR = 2.8), after adjustment for other pesticide exposures.Conclusions: Although we found associations between the risk of NHL and several groupings and specific organophosphate pesticides, larger risks from proxy respondents complicate interpretation. Associations, however, between reported use of diazinon and NHL, particularly diffuse and small lymphocytic lymphoma, among subjects providing direct interviews are not easily discounted. [ABSTRACT FROM AUTHOR]

Published
2001
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47. Increased lymphocyte replicative index following 2,4-dichlorophenoxyacetic acid herbicide exposure.

Author

Figgs, Larry, Holland, Nina, Rothman, Nathanial, Zahm, Shelia, Tarone, Robert, Hill, Robert, Vogt, Robert, Smith, Martyn, Boysen, Cathy, Holmes, Frederick, VanDyck, Karen, Blair, Aaron, Figgs, L W, Holland, N T, Rothmann, N, Zahm, S H, Tarone, R E, Hill, R, Vogt, R F, and Smith, M T

Subjects
CELL nuclei, COMPARATIVE studies, ALCOHOL drinking, GAS chromatography, HERBICIDES, IMMUNOLOGY technique, MASS spectrometry, MEDICAL cooperation, RESEARCH, RESEARCH funding, SMOKING, HYDROXY acids, OCCUPATIONAL hazards, PILOT projects, ENVIRONMENTAL exposure, EVALUATION research, CASE-control method
Abstract

Objective: Evaluate peripheral blood lymphocyte proliferation (replicative index:RI) and micronuclei frequency (MF) among 2,4-D herbicide applicators.Methods: Twelve applicators spraying only 2,4-D provided a blood and urine specimen upon enrollment, several urine samples during the spraying season, and a blood specimen at the study's end. Nine controls provided blood and urine specimens upon enrollment and at the study's end. Gas chromatography/tandem mass spectroscopy determined urinary 2,4-D levels and standard in-vitro assays determined RI and MF scores. Applicator RI and MF were compared before and after spraying and with controls.Results: Applicators contributed 45 urine specimens with concentrations ranging from 1.0 to 1700 (microg 2,4-D/g creatinine/L urine) that logarithmically (In) increased as spraying time increased. Applicator RI increased after spraying (p = 0.016), independent of tobacco and alcohol use, and demonstrated a weak dose-response with increasing urinary 2,4-D levels (p = 0.15). Among 2,4-D applicators, pre-exposure complete blood counts and lymphocyte immunophenotypes were not significantly different from post-exposure measurements.Conclusion: Urinary 2,4-D concentration, an exposure biomarker, may be associated with lymphocyte replicative index, a cell proliferation biomarker. [ABSTRACT FROM AUTHOR]

Published
2000
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49. Cancer incidence and mortality among beta-naphthylamine and benzidine dye workers in Moscow.

Author

BULBULYAN, MARIANA A, FIGGS, LARRY W, ZAHM, SHEILA HOAR, SAVITSKAYA, TATYANA, GOLDFARB, ALEXANDER, ASTASHEVSKY, SERGEI, ZARIDZE, DAVID, Bulbulyan, M A, Figgs, L W, Zahm, S H, Savitskaya, T, Goldfarb, A, Astashevsky, S, and Zaridze, D

Subjects
AMINES, BIPHENYL compounds, BLADDER tumors, DYES & dyeing, LONGITUDINAL method, SEX distribution, TIME, TUMORS, OCCUPATIONAL hazards, ENVIRONMENTAL exposure, DISEASE incidence
Abstract

Background: Cancer incidence and mortality were evaluated among 4581 aniline dye production workers in Moscow.Methods: A historical cohort was assembled and followed-up from 1 January 1975 to 31 December 1989. Moscow district oncologic dispensary registries furnished case ascertainment and employer records provided job exposure data. Expected cancers and deaths were calculated based on gender-, age-, and calendar time-specific incidence and mortality rates for the Moscow general population applied to the cohort's person-years of follow-up. Disease-specific standardized mortality and incidence values were derived from ratios of observed to expected cancers.Results: Men experienced elevated total cancer mortality (standardized mortality ratio [SMR] = 125; 95% CI: 110-142) and urinary bladder cancer mortality (SMR = 279; 95% CI: 192-391), and increased all malignancy (standardized incidence ratio [SIR] = 142; 95% CI: 125-160), oesophageal (SIR = 203; 95% CI: 108-347), respiratory tract (SIR = 154; 95% CI: 120-194) and bladder (SIR = 394; 95% CI: 268-559) cancer incidence. Women had elevated oesophageal (SMR = 313; 95% CI: 124-664) and bladder (SMR = 311; 95% CI: 149-571) cancer mortality and elevated all malignancy (SIR = 124; 95% CI: 106-144), oesophageal (SIR = 348; 95% CI: 140-719), and bladder (SIR = 861; 95% CI: 458-8002) cancer incidence. Bladder cancer rate increased with employment duration and younger age first hired. Rate estimates were highest among beta-naphthylamine exposed workers but was also increased among workers with other chemical exposures. A cancer prevention and control effort that limited benzidine exposure to < or = 3 years was apparently unsuccessful as indicated by a significant excess of bladder cancer (SIR = 1773; 95% CI: 356-5180) among these workers.Conclusion: Relative rates of oesophageal, lung, and stomach cancer were also elevated among all workers, but did not increase with total years worked, age first hired, or year first hired, suggesting a non-occupational aetiology. [ABSTRACT FROM AUTHOR]

Published
1995
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