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1. Joint single-cell genetic and transcriptomic analysis reveal pre-malignant SCP-like subclones in human neuroblastoma

2. Multimodal classification of molecular subtypes in pediatric acute lymphoblastic leukemia

7. Multimodal classification of molecular subtypes in pediatric acute lymphoblastic leukemia

10. Whole‐Genome Sequencing of Cytogenetically Balanced Chromosome Translocations Identifies Potentially Pathological Gene Disruptions and Highlights the Importance of Microhomology in the Mechanism of Formation

11. The clinical impact of IKZF1 deletions in paediatric B-cell precursor acute lymphoblastic leukaemia is independent of minimal residual disease stratification in Nordic Society for Paediatric Haematology and Oncology treatment protocols used between 1992 and 2013

16. Early activating somatic PIK3CA mutations promote ectopic muscle development and upper limb overgrowth

17. Microdeletion of 7p12.1p13, including IKZF1, causes intellectual impairment, overgrowth, and susceptibility to leukaemia

18. A somatic UBA2variant preceded ETV6-RUNX1in the concordant BCP-ALL of monozygotic twins

19. Overexpression of chromatin remodeling and tyrosine kinase genes in iAMP21-positive acute lymphoblastic leukemia

20. Early activating somatic PIK3CA mutations promote ectopic muscle development and upper limb overgrowth

23. High-resolution detection of chromosomal rearrangements in leukemias through mate pair whole genome sequencing

24. Overexpression of chromatin remodeling and tyrosine kinase genes in iAMP21-positive acute lymphoblastic leukemia.

25. Microdeletion of 7p12.1p13, including IKZF 1 , causes intellectual impairment, overgrowth, and susceptibility to leukaemia

26. High-resolution detection of chromosomal rearrangements in leukemias through mate pair whole genome sequencing

27. Whole-Genome Sequencing of Cytogenetically Balanced Chromosome Translocations Identifies Potentially Pathological Gene Disruptions and Highlights the Importance of Microhomology in the Mechanism of Formation

28. PAX5-ESRRB is a recurrent fusion gene in B-cell precursor pediatric acute lymphoblastic leukemia

29. Transcription-coupled genetic instability marks acute lymphoblastic leukemia structural variation hotspots

30. Author response: Transcription-coupled genetic instability marks acute lymphoblastic leukemia structural variation hotspots

31. Detailed gene dose analysis reveals recurrent focal gene deletions in pediatric B-cell precursor acute lymphoblastic leukemia

32. De Novo Loss-of-Function Mutations in USP9X Cause a Female-Specific Recognizable Syndrome with Developmental Delay and Congenital Malformations

33. DNA methylation-based subtype prediction for pediatric acute lymphoblastic leukemia

34. DNA methylation-based subtype prediction for pediatric acute lymphoblastic leukemia

35. DNA Methylation-Based Subtype Prediction for Pediatric Acute Lymphoblastic Leukemia (ALL)

37. DNA Methylation-Based Subtype Prediction for Pediatric Acute Lymphoblastic Leukemia (ALL)

39. Silencing of CEBPB-AS1 modulates CEBPB expression and resensitizes BRAF-inhibitor resistant melanoma cells to vemurafenib

40. Microdeletion of 7p12.1p13, including IKZF1, causes intellectual impairment, overgrowth, and susceptibility to leukaemia.

41. PAX5-ESRRB is a recurrent fusion gene in B-cell precursor pediatric acute lymphoblastic leukemia.

42. Single nucleotide polymorphism genomic arrays analysis of t(8;21) acute myeloid leukemia cells.

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