49 results on '"Yousefzadeh, Matthew J."'
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2. Ending a diagnostic odyssey: Moving from exome to genome to identify cockayne syndrome.
3. PD1 blockade improves survival and CD8+ cytotoxic capacity, without increasing inflammation, during normal microbial experience in old mice
4. Metabolism in the Midwest: research from the Midwest Aging Consortium at the 49th Annual Meeting of the American Aging Association
5. Neurodegeneration as the presenting symptom in 2 adults with xeroderma pigmentosum complementation group F.
6. An aged immune system drives senescence and ageing of solid organs
7. Signal Transduction, Ageing and Disease
8. Sex Makes a Difference in Aging: Biology and Beyond
9. Cellular senescence: a key therapeutic target in aging and diseases
10. Heterochronic parabiosis regulates the extent of cellular senescence in multiple tissues
11. ERCC1-deficient cells and mice are hypersensitive to lipid peroxidation
12. Senescence and Inflammation: Summary of a Gerontological Society of America and National Institute on Aging-Sponsored Symposium
13. PD1 blockade improves survival during lethal microbial exposure in old mice
14. Mouse Models of Accelerated Cellular Senescence
15. Loss of DNA repair mechanisms in cardiac myocytes induce dilated cardiomyopathy
16. ERCC4 variants identified in a cohort of patients with segmental progeroid syndromes
17. Heterochronic parabiosis: a valuable tool to investigate cellular senescence and other hallmarks of aging
18. The Role of DNA Repair in Immunological Diversity: From Molecular Mechanisms to Clinical Ramifications
19. Novel small molecule inhibition of IKK/NF‐κB activation reduces markers of senescence and improves healthspan in mouse models of aging
20. Metabolism in the Midwest: research from the Midwest Aging Consortium at the 49th Annual Meeting of the American Aging Association
21. Case Report: Identification of a Heterozygous XPA c.553C>T Mutation Causing Neurological Impairment in a Case of Xeroderma Pigmentosum Complementation Group A
22. The Second Annual Symposium of the Midwest Aging Consortium: The Future of Aging Research in the Midwestern United States
23. Senolytics reduce coronavirus-related mortality in old mice
24. REV7 is essential for DNA damage tolerance via two REV3L binding sites in mammalian DNA polymerase ζ
25. Exercise reduces circulating biomarkers of cellular senescence in humans
26. Influences of circulatory factors on intervertebral disc aging phenotype
27. ATM is a key driver of NF-κB-dependent DNA-damage-induced senescence, stem cell dysfunction and aging
28. Tissue specificity of senescent cell accumulation during physiologic and accelerated aging of mice
29. Adenoviral gene transfer of a single‐chain IL‐23 induces psoriatic arthritis–like symptoms in NOD mice
30. Measuring biological age in mice using differential mass spectrometry
31. Fisetin is a senotherapeutic that extends health and lifespan
32. Spontaneous DNA damage to the nuclear genome promotes senescence, redox imbalance and aging
33. Adenoviral Gene Transfer of a Single Chain IL-23 Induces Psoriatic Arthritis-Like Symptoms in NOD Mice
34. Circulating levels of monocyte chemoattractant protein‐1 as a potential measure of biological age in mice and frailty in humans
35. Metabolism in the Midwest: research from the Midwest Aging Consortium at the 49thAnnual Meeting of the American Aging Association
36. Analysis of DNA polymerase ν function in meiotic recombination, immunoglobulin class-switching, and DNA damage tolerance
37. Essential Roles for Polymerase θ-Mediated End Joining in the Repair of Chromosome Breaks
38. Circulating levels of monocyte chemoattractant protein‐1 as a potential measure of biological age in mice and frailty in humans.
39. <italic>ERCC4</italic> variants identified in a cohort of patients with segmental progeroid syndromes.
40. Frailty and pre-frailty associated with physical functional performance and quality of life in breast cancer and bone marrow transplant survivors.
41. Mechanism of Suppression of Chromosomal Instability by DNA Polymerase POLQ
42. DNA polymerase POLQ and cellular defense against DNA damage
43. REV7 is essential for DNA damage tolerance via two REV3L binding sites in mammalian DNA polymerase ζ.
44. Cellular senescence: a key therapeutic target in aging and diseases.
45. Adenoviral gene transfer of a single‐chain IL‐23 induces psoriatic arthritis–like symptoms in NOD mice
46. Spontaneous DNA damage to the nuclear genome promotes senescence, redox imbalance and aging
47. PD1 blockade improves survival and CD8 + cytotoxic capacity, without increasing inflammation, during normal microbial experience in old mice.
48. Failure to repair endogenous DNA damage in β-cells causes adult-onset diabetes in mice.
49. Mouse Models of Accelerated Cellular Senescence.
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