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1. Stratification of radiosensitive brain metastases based on an actionable S100A9/RAGE resistance mechanism

2. Mapping molecular subtype specific alterations in breast cancer brain metastases identifies clinically relevant vulnerabilities

3. Comparative analysis of the AIB1 interactome in breast cancer reveals MTA2 as a repressive partner which silences E-Cadherin to promote EMT and associates with a pro-metastatic phenotype

5. A clinically compatible drug‐screening platform based on organotypic cultures identifies vulnerabilities to prevent and treat brain metastasis

7. ADAM22/LGI1 complex as a new actionable target for breast cancer brain metastasis

21. Supplementary Methods from Epigenome-wide SRC-1–Mediated Gene Silencing Represses Cellular Differentiation in Advanced Breast Cancer

22. Supplementary Table S2 from Transcriptomic Profiling of Sequential Tumors from Breast Cancer Patients Provides a Global View of Metastatic Expression Changes Following Endocrine Therapy

23. Figure S2 from BET Inhibition as a Rational Therapeutic Strategy for Invasive Lobular Breast Cancer

24. Supplementary Tables from Epigenome-wide SRC-1–Mediated Gene Silencing Represses Cellular Differentiation in Advanced Breast Cancer

25. Supplementary Table 2 from AIB1:ERα Transcriptional Activity Is Selectively Enhanced in Aromatase Inhibitor–Resistant Breast Cancer Cells

26. Supplementary Figures from Adaptation to AI Therapy in Breast Cancer Can Induce Dynamic Alterations in ER Activity Resulting in Estrogen-Independent Metastatic Tumors

27. Supplementary Table 2 from Brain Metastasis Cell Lines Panel: A Public Resource of Organotropic Cell Lines

28. Table S2 from BET Inhibition as a Rational Therapeutic Strategy for Invasive Lobular Breast Cancer

29. Supplementary Table 3a from AIB1:ERα Transcriptional Activity Is Selectively Enhanced in Aromatase Inhibitor–Resistant Breast Cancer Cells

30. Supplementary Figures from Epigenome-wide SRC-1–Mediated Gene Silencing Represses Cellular Differentiation in Advanced Breast Cancer

32. Figure S1 from Brain Metastasis Cell Lines Panel: A Public Resource of Organotropic Cell Lines

33. Supplementary Figures S1-7 from Transcriptomic Profiling of Sequential Tumors from Breast Cancer Patients Provides a Global View of Metastatic Expression Changes Following Endocrine Therapy

34. Data from Brain Metastasis Cell Lines Panel: A Public Resource of Organotropic Cell Lines

35. Supplementary Table 1 from Brain Metastasis Cell Lines Panel: A Public Resource of Organotropic Cell Lines

36. Supplementary Figures 1 - 9 from AIB1:ERα Transcriptional Activity Is Selectively Enhanced in Aromatase Inhibitor–Resistant Breast Cancer Cells

37. Supplementary Table 1 from AIB1:ERα Transcriptional Activity Is Selectively Enhanced in Aromatase Inhibitor–Resistant Breast Cancer Cells

38. Supplementary Tables from Adaptation to AI Therapy in Breast Cancer Can Induce Dynamic Alterations in ER Activity Resulting in Estrogen-Independent Metastatic Tumors

49. Steroid Ligands, the Forgotten Triggers of Nuclear Receptor Action; Implications for Acquired Resistance to Endocrine Therapy

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